Featured Story

NEW LYME BOOK: The most important one ever published?

Burton Waisbren Lyme Book
Treatment of Chronic Lyme Disease: Fifty-One Case Reports and Essays in Their Regard


Paperback Book, $24.95
Published in January, 2012

This book is quite possibly the most important Lyme disease book ever published. It was written by one of the Founding Members of the Infectious Diseases Society of America, an organization which has denied the legitimacy of chronic Lyme disease. Not only does Dr. Waisbren bring his credibility to the table in asserting the reality of chronic Lyme disease, he also shares with the reader 51 cases of the disease which he has personally treated, along with the many treatment methodologies he uses in his Lyme disease practice. He has been practicing medicine for over 57 years and has published Lyme disease research in many journals, such as The Lancet. 

About BioMed

BioMed Publishing Group was founded in 2004 by Bryan Rosner. The company exists to educate the world about Lyme disease, tick-borne illness, and related health topics. As a publishing company, we are always looking for new authors with original book ideas.


BioMed Publishing Group hosts two Lyme-related forums. Membership in the forums is easy and free! Start participating in the discussion today:

  1. Lyme Community Forums
  2. The Lyme/Rife Discussion Group

If you have questions, please contact BioMed Publishing Group.

Submit a Book Proposal

Are you ready to publish information on Lyme Disease, alternative medicine, or a related topic? BioMed Publishing Group is the leader in providing useful and informative printed and digital informational resources in the arena of tick-borne disease, alternative healthcare, and topics of interest in similar genres. We are always looking for authors who bring new, interesting ideas to our line of products. If you have a book idea or a completed manuscript, we would like to hear about it. Please submit your book proposal here.

Guest Blog post – FIR Saunas

This is a guest blog post from one of our sponsors, Heavenly Heat Sauna. Some great information!



Body heating has been used for centuries for pain and stiffness, body purification, relaxation, rejuvenation, and more recently, as a holistic, adjunctive modality for treating autism, cancer and Lyme disease. Saunas can be powerfully effective by increasing blood flow, raising body temperature, and inducing profuse sweating to excrete toxic chemicals and heavy metals.

A good far-infrared (FIR) sauna can increase blood flow and induce sweating more profoundly than in a considerably hotter traditional sauna. This occurs due to the greater speed with which infrared energy – a light wave, warms the body as compared to hot air. This, by the way, has nothing to do with any “special wavelengths” that some promoters falsely claim make their saunas uniquely more effective than others.

There are two factors in sauna designs that really matter for efficacy. The first is to create optimal infrared exposure levels, and the second is to construct the sauna to create a most healthful detoxification environment.

Effective IR levels are achieved by using ceramic heaters rather than carbon or near-infrared bulbs. Carbons are typically made of covered plastic and are weak, making these saunas effective only at higher temperatures. NIR is absorbed by the body in a less concentrated manner than FIR and this makes it less effective for detoxification, and it may create optical risks if it’s not positioned properly.

Heavenly Heat creates ideal sauna detox environments. Each sauna is built entirely with low-allergen white poplar wood and tempered safety glass, without the use of plywood, laminates, or any wood glue. A constant air-change is built into each model.

FIR can enhance and accelerate the sauna detox process, and can be highly beneficial but only if the sauna is designed and built properly. That is a big “if” as the marketplace is flooded with Asian imports that are built improperly for a detox environment with strong adhesives and plastic heaters.

Heavenly Heat’s saunas can be viewed at www.heavenlyheatsaunas.com and brochures with pricing and specifications can be requested at info@heavenlyheatsaunas.com or by calling 800-697-2862.

Research Shows Stevia Is Effective Against Lyme Disease

stevia_high_1933_generalStevia leaf continues to be studied for it’s anti-Lyme activity. Research is increasingly pointing to Stevia as a useful Lyme therapy.

But different brands of Stevia appear to vary substantially in how useful they are, so be careful which one you use. The below-linked study compared 5 different brands of Stevia, and found that the Nutramedix brand is the most effective (represented in the study as “Stevia A”). Very interesting study to read, if you have time, check it out.

It is also interesting to see that some brands do absolutely nothing; so be sure you don’t discount this treatment until you’ve tried one of the effective brands.

Read the full study text:


Bryan Rosner entering Mr. Universe competition

IMG_1377Am I posting this picture of myself because I think I can enter a Mr. Universe contest (not!) or because I am trying to teach you something about staying healthy when you are recovering from Lyme disease, especially during the holiday season? Subscribe to my Anti-Lyme Journal to find out! Learn more by clicking here…

Food & Inflammation – the final frontier in Lyme disease treatment

It’s been over 10 years since I wrote my first Lyme disease book. In 2014, I completed my last book, Freedom From Lyme Disease.

Now, in 2015, I’m reflecting on the final frontier in Lyme disease treatment. As it is very difficult to COMPLETELY eradicate the bacteria, we are faced with controlling the final symptoms after 99% of the bacteria are removed or under control.

As it turns out, the two areas with a very high degree of importance in this time frame are FOOD and INFLAMMATION.

During this final healing period, food reactions become much more pronounced. Food sensitivities – as well as inflammatory foods like sugar – have an exaggerated effect as the body is desperately attempting to maintain equilibrium (and now for the first time, can actually have a chance at doing so). Therefore, the goal in the last phase of healing should be strict control of the diet, eliminating most sugars and greatly limiting fruit, as well as trying to eliminate ALL food sensitivities including gluten, most grains, and other foods a person can be allergic to. This can literally be the make or break in the Lyme disease end game, as food reactions can throw a person into old Lyme symptoms while a clean diet can allow one to feel normal.

In contrast, early on in the recovery process, food reactions and sugar made things worse but the body was too overwhelmed to really notice, so the results of eating a good – or bad – diet didn’t really make much of a difference.

Remember, the key elements here are: avoid sugar (even fruit sugar) as much as possible, avoid carbohydrates as much as possible, and avoid all food sensitivities including gluten and other personal allergies.

The next area to consider, then, in the final phases of healing, is inflammation. Inflammation can remain at a low grade long after most bacteria are gone, and it can allow depression, anxiety, brain fog, fatigue, and other symptoms to persist. For inflammation, use the diet guidelines above but also consider herbs like curcumin, nettles, tart cherry, and others.

If you listen to your body and control your diet and inflammation, along with using the rotation protocol described in my books, you should be able to maintain a very low level of symptoms during the final phases of healing.

Claritin kills Lyme bacteria! Must read!

Researchers at Stanford have now shown that Claritin, i.e. loratadine, kills Borrelia. Read more here.

FREE CHAPTER from my new book! (From Bryan Rosner)

Hello everyone,

Here’s a FREE CHAPTER from my new Lyme disease book. Enjoy!


GUEST BLOG: My Life in the Lyme Light

My Life in the Lyme Light                  

A city girl from Denver learns the source of her chronic suffering…

and discovers that Lyme disease can strike anywhere

By Linda Warner, guest blogger

Let me start by saying that I am an overcomer of many things.   I grew up in a very abusive alcoholic home, where my parents struggled to put food on the table.  I left my home at 16 to enter an abusive relationship with my daughter’s father.  I stayed in the relationship far too long, but knew it was not what God’s plan was for me.  I put myself through school, even going on to get an MBA.  I ended that relationship.  But nothing has been more draining to overcome the Diagnosis of LYME.    I know that God has given me the strength and the tools to overcome anything.

I am not sure when I got bitten by a tick. I experienced no bulls eye rash. What I did experience, seemingly from out of the blue, was bone-deep achiness and endless fatigue and anxiety.   Psychiatrists diagnosed me with everything from PTSD to BiPolar.

As a single parent, marathoner and hard driving, middle-aged sales rep for a major global pharmaceutical company, I told myself this was normal. After all, my work had me in and out of doctors’ waiting rooms all day long — of course I would be prone to picking up whatever the patients were bringing in. Plus, wasn’t this what all “women of a certain age” go through?

One fall day in 1989, driving to a sales call, I literally fell asleep at the wheel. I awoke after colliding with a tow truck. While the truck driver was fine, I suffered a back injury and my body ached worse than ever.

The accident really scared me and served as a virtual “wake up” call to get help.  Surrounded by doctors at my job, I had easy access to a wealth of professional advice and diagnoses. And everybody pretty much said the same thing: It was all stress and anxiety.


Thus began a cycle that always began with a deep pressure behind my eyeballs. I’d visit my doctor, who would diagnose a sinus infection – never mind that my nasal passages were absolutely clear. Within a few days on antibiotics, the pressure would lift and I would feel better. But soon the symptoms would reappear, and the cycle would begin again.

And that’s the way it was day after day. It was like that tow truck kept hitting me again and again. I popped antibiotics and antidepressants like candy. They were my lifeline, lifting the pain and anxiety and weariness just enough to keep me going.

In 2005, attracted to Colorado’s sunshine and clean air, I moved my family to Denver. After a short hiatus, I took a new job. I was still calling on doctors, but this time they were integrative and holistic, using many different kinds of nontraditional therapies. It was here that I learned about neurotransmitter testing.

As a pharmaceutical rep, I already knew that neurotransmitters are brain chemicals that act as messengers in the body, and that they are required for proper brain and body functions. From these new integrative physicians, I learned that since physical and psychological challenges can cause variations in neurotransmitter levels, it’s helpful to measure them.

Holding out little hope, I agreed to neurotransmitter testing. My results came back high for inflammation, glutamate and GABA. As a result, my doctor urged that I start an anti-inflammatory diet, which I expected…but I never expected she’d also recommend additional testing for Lyme.  After all, that’s something campers in Wisconsin get, right? I spend most of my time in my car in Colorado!

Needless to say, I was skeptical. But I was also sick and tired of being sick and tired. So I agreed to what my doctor called a “one-two punch”: traditional Western Blot testing, which detects Lyme antibodies, plus a new test called iSpot Lyme, which measures Lyme antigen-specific T cells. By testing for both antibodies and T cells, she told me, there would be a better chance of finding where Lyme might hide. (Full disclosure: iSpot Lyme was developed by a lab affiliated with my employer.)

 My results: I was definitely positive for Lyme, with my iSpot Lyme test showing over five times the limit for a positive diagnosis. Under my doctor’s care, I immediately began aggressive, targeted treatment. I have been through many courses of Antibiotics, and other medications and herbs to address the inflammation, biofilms, candida parasites and co-infections.  I have recently been adding frequency treatments.

Recently, I was retested. Although the Lyme bacteria are still present, they are now at much lower levels. Better yet, the deep achiness and fatigue have subsided and I am nearly off my medications. I am beginning to get my life back. I enjoy spending time with my teen children and grandbaby.

I am learning to support my immune system and pray I can find full remission of this dreadful disease.

Where did a city girl from Denver get Lyme? It’s hard to say, but as I trace back my symptoms, I recall they started shortly after a business trip to Tennessee. After three weeks of sales training, I unwound with a leisurely bike ride through a cool, deep forest. Who knew that what should have been a relaxing end to a long business trip would be the beginning of a quarter-century search for the source of my pain?

Through this journey, I have become a passionate advocate for Lyme disease testing.  It’s a common misconception that if you don’t live in New England or the Upper Midwest, you can’t get Lyme. Today, everyone is so mobile that even if you are fortunate enough to live in a place with few ticks, you probably still visit places that do. And all it takes is a single bite from a critter smaller than a poppy seed.

Lyme mimics symptoms of other diseases, so it’s hard to pin down. Like me, if you don’t get the famous bulls eye rash, most doctors won’t diagnose it. And, like me, if you’re diagnosed incorrectly, it can lead to years of suffering.  The symptoms that proved most bothersome for me were gut wrenching anxiety, fatigue , brain fog and achyness

The best way to be sure is to get tested. It can give you peace of mind knowing whether you are positive or not …wherever you call home.


Biofilm Treatments for Lyme

Watch the video:

Combating Biofilms Book – By James Schaller, MD – Just Released April, 2014!

Dr. James Schaller’s new book is now here! Get your copy of Combating Biofilms book today!


Click here to buy your copy for $29.95

Who Has Biofilm Infections?

When you learn about the massive diversity of locations and situations in which biofilms are common and consider that that are often the routine state of bacteria and fungal organisms, you start to realize anyone may have a biofilm infection or infections.

What Are We Looking For in This Book?

The following material will show many ways to break through the “egg white,” or biofilm. Once that happens, it is usually much easier to destroy the infection represented by the egg yolk or yellow center.

Biofilms Are a Leading Cause of Suffering and Death

Biofilm Body Locations and Situations

• An infection lasting over 2 weeks

• The leading cause of death in children under 6 years of age

• Dental plaque—the human mouth harbors about 25,000 species of bacteria, about 1,000 of which reside in the dental plaque biofilm.

• Yeast infections

• Postsurgical infections

• Cancer

• Bad breath

• Gum disease or periodontitis*

• Tooth decay

• Lung infections

• Urinary system infections

• Oral bacteria—can harm heart arteries and cause death and increase intestinal cancers

• Chronic ear infections

• Sinus infections**

• Chronic tonsillitis

• Wounds

• Tooth brush heads — including sonic moving head styles

Catheters to allow urine removal

• Artificial knees, hips, and other replacements

• Heart valve infections

• Lesions or sores

• Lyme disease

• IV catheters of any type

• Urinary catheters

• Contact lenses

• Implanted devices—any implanted or inserted device can send bacteria to the brain, liver or kidneys.

• Chronic prostate infections

• Legionnaire’s disease and many other biotoxin bacteria that explode in any indoor water

• Mold illnesses—which can arise from mold build up in any standing indoor water, i.e., flooding, roof, basement or window leaks, humidifiers, unused Waterpik™ or other tooth cleaning devices, condensation in AC ducts, etc.

• Cystic fibrosis—excess mucus production in the airways allows bacteria like Pseudomonas aeruginosa to beat bacteria killers behind a biofilm coat.

• Lost body parts

• Skin, hair or nail infections

• Arthritis

• Endocarditis

• Bone infections

• Acne

Many other things could be added to the list, including profoundly serious issues of biofilm contamination in water and dozens of other health-related and manufacturing practices.

*Doctor David Kennedy, a retired dentist, lamented that most adult Americans have gum disease—another bacterial biofilm condition involving chronic infection. So just how widespread is this stealthy healthcare epidemic?

**At Ondine Biopharma, an interview [with Richard Longland] revealed that 38,000,000 people in this country have (or had) a chronic sinus problem.

***Ricardo Murga; Terri S. Forster. Role of biofilms in the survival of Legionella pneumophila in a model potable-water system. Microbiology (2001), 147, 3121–3126.

 A Medical Revolution

The theory of biofilm infection is a profound revolution in the study of infections which can be painful, disabling and in fact, are a top killer depending on one’s age. Infections are starting to return us to the days when people died of simple infections. The new biofilm infection world could kill more people than WWI and WWII combined if things do not quickly change in both developed and undeveloped nations. Due to a slow understanding of the importance of biofilms and therefore, a slow adoption by physicians of new biofilm solutions, even cutting edge doctors might only take biofilms seriously when it has been proven that more people are becoming disabled and die due to them. Currently, most miss biofilms as the cause of suffering and death.

So, biofilms without solutions are as serious as polio in the 19th century without a vaccine, and in terms of numbers of victims, they are far more devastating than HIV/AIDS. Most bacteria live in communities that typically have unique protective biofilms. 1% of bacteria infecting humans or impacting human life are floating alone and when they are found in blood, they would not be found together with any biofilm slime. The National Institutes of Health estimates that more than 80% of microbial infections in the human body are caused by biofilm, many of them creating chronic and reoccurring problems. Or, is Glowacki right and 99% of bacteria live in a biofilm? Whether you use NIH’s 80% or Glowacki’s 99% as the estimate, biofilms are a serious consideration in infections. Głowacki R, Strek P, Zagórska-Swiezy K, Składzień J, Oleś K, Hydzik-Sobocińska K,Miodoński A. [Biofilm from patients with chronic rhinosinusitis. Morphological SEM studies].[Article in Polish]. Otolaryngol Pol. 2008;62(3):305-10.

 Making Biofilms Clear

Making “Biofilms” Clear A biofilm is like a dime in the center of a pool of olive oil, and on the outer edge of the oil is pepper representing infection killing cells. They cannot move in to destroy the dime. Biofilm bacteria communities are the usual state of most human infections. We have been taught that infections are isolated bacteria floating around and this is a serious error. It shows how far we need to go in science if the main form of bacteria— biofilm bacteria communities—is a new, but crucial, concept. When I made a list in 2004 of twenty-five options to kill biofilms, there wasn’t much interest. The goal in writing and publishing this book is to make an affordable research- based set of options along with other possible options, to present a pure book of solutions offering the newest possible current and up to date solutions for the hundreds of diseases associated with biofilms. The barrier of a biological film can be utterly impossible to remove or penetrate with the routine options used by physicians, infection specialists, naturopaths, alternative medicine schools, essential oil practitioners, acupuncturists, nurse practitioners or herbalists.

With this book we hope to serve you and your physician/healer by the exploration of options available now. We searched the past five years of publications on PubMed—the massive database for medical science— for “biofilm treatment.” The range of options is impressive and not always things you might expect. This book is meant to give you broad options to prevent your suffering, disability and even death. After years of research and study, I have come to realize that the infectious disease “experts” on biofilm may have long since lost the war, and in fact, many may not ever have been aware of all the battles. Patients and researchers were learning basic things about infections in 2012 and 2013 which shatter trust in infection specialists.

Most people, and infectious disease doctors themselves believe that an infection physician knows all infections. Much of their work is related to HIV, Hepatitis B and C, Tuberculosis, strong pneumonia, the antibiotic resistant staph infection MRSA, sepsis [bacteria in the blood], post surgical infections, Flu, Meningitis, Rotavirus, Streptococcus, Clostridium difficile (caused in part by lack of knowledge of the benefits of high quality probiotics), infections of various body part implants, and a finite number of other serious infections. Many of these are very hard to treat, such as AIDS, which requires the care of someone trained in very advanced medical science.

The point is, however, that infection physicians do not usually have the time to research all the options to handle biofilms because even to become an infection “expert” on just one infection, it takes about a year to read all the applicable articles, ponder, and see how they may apply to many people. Therefore, our goal is to advance this area involving all healers and millions of patients. Most traditional healers are limited by the options of pharmaceutical companies. I have appreciated receiving small grants awarded by some of these companies in the past. They have given me grants knowing the resulting information published was outside their control. It is not true that all synthetic medications are bad, however, some can be dangerous to use and some can be far worse options than those used in functional medicine or by integrative physicians.

On the other hand, the situation could be better if the education on effective dosing, delivery flexibility and risks of many alternative treatments, herbs, etc. were better. Starting with hospitals and traditional medicine, the current approach of removing biofilms using another antibiotic or other patented synthetic agent will fail today or next year in many or some cases. Profoundly unique agents that block signals between the bacteria involved in making the biofilm “fort,” or the use of virus carriers to attack some part of the biofilm, will likely be introduced soon, along with dozens of other advanced options. Much to our detriment, we do not have them now. The FDA approval hurdle is massive and takes many years.

The current treatments offered in integrative, alternative or functional medicine are often too simplistic, but some useful ones you may see are available today. At the other extreme, routine allopathic MD medicine seems to feel that only prestigious schools have solutions right now from multi-million dollar studies—they do not. Even some patients with biofilm infections who are very sick or dying in many traditional hospitals or under routine medical care are frankly “left by the side of the road” to suffer, become disabled or die. Alternative or functional medicine options often result from very poor reasoning based on limited information. The most common trouble might be that treatment “d” or “e” or a mix of treatments “abcdefghijklm” helps Tom or Ann, so they are excited. They have found gold in the backyard, and in their sincere joy, they post this information. When research assistants follow up with these patients in one to three years, they are typically worse. Smart physicians and other healers of any philosophy or training do not appreciate that destroying pathological biofilms is like trying to open a steel door using a banana as the key to a two ton lock.

The limited treatment and supposed cures of many physicians and healers trying to remove biofilms did not take place when checked by long term follow up.

Free interview with Bryan Rosner & Dr. Horowitz when you buy his book from us!

If you haven’t already, check out Dr. Horowitz’s new book, Why Can’t I Get Better:


When you order from us, you get a FREE downloadable interview between Bryan Rosner & Dr. Horowitz. Click above to learn more.

Custom Probiotics at 15% off…

One of my favorite probiotic companies is having a 15% off sale this month… if you haven’t used their products before, I suggest you try them.



Note: Custom Probiotics sponsors Lymebook.com.

Bryan Rosner talks to Connie Strasheim in audio interview

Did you know that you get a FREE audio interview between Bryan Rosner and Connie Strasheim when you purchase Connie’s new book, Beyond Lyme Disease, directly from our website? The interview includes lots of cutting-edge new information, and discussion of Connie’s new book. The book is now one of our bestsellers as many Lyme sufferers realize that in order to get well, one must address more than just infections. Check it out today and read a free chapter on our website!


New Details on Groundbreaking Book by Dr. Richard Horowitz

Why Can't I Get Better By Richard Horowitz

I am getting exclusive new details on the release of Dr. Horowitz new Lyme disease book. The release date is slated for Sept 3, 2013, and the title is now looking firm as:

Why Can’t I Get Better? Solving the Mystery of Lyme and Chronic Disease

Click on the above link for all the new details, including his speaking schedule for 2013, a detailed description of the book, and more! On the page, you can also sign up to be notified of this book’s release.

Why Can’t I Get Better By Richard Horowitz


2012 ILADS meeting summary… must read!

If you read one thing this month, read this: BetterHealthGuy’s fantastic summary of the 2012 ILADS meeting. I love these summaries–I get to digest a good portion of the information without spending the time or money to attend the event. Thanks, Scott!


Also, we now have information on the 2013 ILADS meeting.  It will be held in Hershey, PA, April 19-20, 2013.  To learn more or register, visit: http://ilads.org/ilads_media/hershey-registration/


Dr. Horowitz’s new book now has a title

Dr. Horowitz, who has treated more than 10,000 Lyme disease patients and is regarded as one of the top LLMD’s in the world, has announced the title of his new book. The title was changed recently; prior, he had announced it as World Out of Balalnce. Now, it appears, based on a recent ILADS newsletter, that the title of his new book will be:

Why Can’t I Get Better? Solving the Mystery of Lyme and Chronic Disease

We expect the book to be available sometime in Summer, 2013. To get notified when it is published, click the above link, then click on, “get notified.”



Decoding The Mystery of Chronic Illness – Conference DVD Sets Available

DVD Sets Now Available From Recent Conferences

Dennis Schoen at Researched Nutritionals has sponsored two very informative conferences; now you can get your DVD sets from these conferences, and save $35 when you buy both sets together. Learn more here.

Topics include biofilm, coagulation, and inflammation, and Dr. Burrascano is one of the speakers.

One of the conferences was held in September, 2012 and the other in April, 2011.

Fantastic information on the Cowden Protocol

This is a great, central source of free information on the very important Cowden Protocol:


I found the videos especially useful. Enjoy!

Amazon discounting new Lyme book with free shipping!

Hurry! If you’ve been waiting for the right time to buy Nicola’s new Lyme Disease book, now might be that time: Amazon is currently discounting it to $26.37 with free shipping.


New Lyme Disease book by Connie Strasheim

The new book is entitled Beyond Lyme Disease. Learn more about the book here. Watch Connie talk about the book, below.


Liposomal supplements … a breakthrough you need to know about!

Many people have heard of “liposomal” supplements but wonder if they are hype, or for real. They are for real. Liposomal vitamin C has been shown to be absorbed up to 5x as effectively as any other type of vitamin c. Liposomal products are encapsulated in a fatty matrix which makes them much more fat-soluble and readily absorbed from the intestinal tract. One of my favorite liposomal vitamin c brands is Liv-On Laboratories Liposomal Vitamin C, which can be purchased from Amazon.com or iHerb.com.

Researched Nutritionals* also makes a liposomal glutathione product. Glutathione is the body’s “master antioxidant” and the liposomal form of this product gets the substance where you need it, faster, bypassing the process of digestion.

Recently, Hopkinton Drug* released a liposomal artemisinin product which appears to be highly promising. Hopkinton Drug is also one of the only suppliers in the country which, at the present time, has in stock the important anti-parasitic herb known as Mimosa Pudica and recommended by Dr. K. Remember: these antiparasite treatments aren’t just for helping deworm the gut, they also help break down biofilm throughout the body and can lead to dramatic symptom improvement. In fact, one well-known Lyme doctor has documented worm DNA in Lyme biofilms, indicating that the action of these antiparasitic herbs and drugs reaches far beyond the gut.

Hopkinton Drug’s Mimosa Pudica and Liposomal Artemisinin are over-the-counter and can be ordered by calling: (800) 439-4441.

Also, don’t miss this flyer from Hopkinton Drug with more information on their products. Don’t miss the back side of the flyer, page 2, with information on Mimosa Pudica and Liposomal Artemisinin.

* Disclosure: Lymebook.com is sponsored by Hopkinton Drug.

* Disclosure: Lymebook.com is sponsored by Researched Nutritionals.

Q & A with Dr. Cowden

Damaged Rife Handbook available & recent Lyme conference notes

If you have been thinking about picking up Nenah Sylver’s 2011 Rife handbook, which retails for $112.50 and is about $122.50 including shipping, I have a slightly damaged, brand new copy available that I’ll sell for $65 which includes shipping. The spine has a dent in it but the book is completely usable. If interested contact me at bmpublish [at] gmail.com. One copy, so first come, first served.

And just so those of you who aren’t interested in this book don’t feel like they are wasting their time reading this, here is a very good summary of a recent Lyme conference. Enjoy.


Dr. Burrascano: “Putting Lyme Behind You” 2-DVD Set

Let Dr. Burrascano tell you how to “Put Lyme behind you.” A 2-DVD set brought to you by LymeDisease.org (formerly CALDA) and Gordon Medical Associates. Buying this special DVD set from us will also get you the special bonus: a 1-year FREE membership to LymeDisease.org, featuring the quarterly “Lyme Times” periodical mailed to your doorstep, a member’s only area, and many other benefits. Order your copy of this DVD set today or learn more here.

2-DVD Set • $50

2-DVD Set • $50

Learn more about the DVD set

Cowden Support Program Workshop: May 17, Klagenfurt, Austria

For those of you who live in Europe and want to learn more about the Cowden Support Program:

Nutramedix is hosting the 

Cowden Support Program Workshop- May 17, Klagenfurt, Austria

 ·         Lee Cowden, MD, will discuss program details and present clinical and in vitro study results

 ·         Carsten Nicklaus, MD, PhD will present the clinical results of a clinical study recently conducted at the Borreliose Centrum Augsburg

 ·         Armin Schwarzbach, MD, PhD will explain laboratory tests from patients that participated in the study at BCA

 ·         Click here for more information about the workshop

 ·         Nutramedix will provide one free 6-month CSP (patient cost1,476 EUR) to each licensed practitioner that attends

 ·         To register for the workshop contact Nutramedix atinfo@nutramedix.com

 ·         Click here for more information about the Cowden Support Program

  Nutramedix is also exhibiting at the

 3rd International Lyme & Associated Diseases Society (ILADS) European Meeting- May 18-19Klagenfurt, Austria


  ·         Click here for information about the meeting



Mimosa Pudica, herb described by Dr. Klinghardt, is back in stock!

Mimosa PudicaMany people are now aware of Mimosa Pudica, an ayurvedic herb recommended by Dr. Klinghardt for parasites and possibly Babeisa.

According to Scott Forsgren’s notes on the Deep Look Beyond Lyme conference which took place in 2011, Dr. Klinghardt says that Mimosa Pudica is 30 times stronger than the best pharmaceutical drug.

The problem has been getting the stuff. Various reports across the web indicate that some sources may be low-quality and cause side effects or sickness. The most reliable source, according to some, is Hopkinton Drug, a compounding pharmacy in Massachusetts. However, due to high demand, this pharmacy had been out of stock for several months. Yesterday I spoke with someone at the pharmacy and I am pleased to report that the product is back in stock. Furthermore, it is available OTC (over the counter) at a price which is much lower than I’ve previously seen. You can contact Hopkinton Drug at (508) 435-4441 ext. 117 or ext. 118, or email them at support@rxandhealth.com.

Related: Don’t miss Hopkinton Drug’s Top 3 Lyme Disease Formulations (PDF flyer).

There have been various studies done on this species of plant and its various effects, including studies on antimicrobial effects, and anti-anxiety effects.  Please note, though, that this product is still experimental and that conclusive results about its usefulness and safety are still being evaluated by LLMDs.

Does the herb work? I’m in touch with one person who says they feel much better when taking it. However, I think it is too early to draw conclusions. I hope people report their results on this blog, so we can compile some user experiences.

If you use this product, please report your results here on this blog, so that others can learn from your experience. Also, please only use this product under the supervision of a licensed physician. For dosing information, please consult a physician who is experienced with using Mimosa Pudica. DISCLOSURE: Hopkington Drug is an advertising partner with BioMed Publishing Group, the publisher of this blog.


3 members of congress on chronic Lyme disease

Don’t miss this letter written a week ago by 3 members of congress, denouncing the old chronic Lyme guidelines which do not acknowledge the existence of chronic Lyme disease:


The authors of the letter are Chris Smith, Frank Wolf, and Chris Gibson, all members of United States Congress.

California government finally recognizes Lyme


CDPH Cautions Californians About Ticks And Tick-Borne Diseases


Dr. Ron Chapman, director of the California Department of Public Health (CDPH) and state public health officer, today warned individuals who work or play outdoors in the winter months to be on the alert for ticks that may carry bacteria that cause Lyme disease and other tick-borne illnesses.



A sad day … the passing of Jim Meissner

Jim Meissner was actively involved in rife research for the last several years and I believe, made some significant contributions to our community. (NOTE: Please consult a doctor before experimenting with rife therapy. Jim Meissner was not a doctor).


This is to inform members of this list of the passing away of Jim Meissner at the age of 70 from cancer.  He is a member of this group

(though hasn’t posted much for several years I believe).  He was an inventor and sold a CS generator among other things.  He first introduced me to silver which did in fact save my life, and I believe is still what is keeping me alive.  The last several years of his life were largely spent focusing on people with lyme disease and its coinfections.  He invented a number of rife-type devices which have profoundly benefited or even remitted a number of people with lyme-related illness.  His forum on this topic will continue to be carried by others.  He pursued alternative treatments, and got significant results, but it seems the diagnosis may have just come too late.  6 days before his passing, he willfully decided to stop the fight.  He suffered no pain except the last 4 days of his life after ceasing the use of his rife-type devices. 

Below is a copy of his local obituary: 

Juergen “Jim” Paul Meissner 

Juergen “Jim” Paul Meissner, 70, died Tuesday, December 20, 2011 of colon cancer at his residence in Afton.  He was preceded in death by his parents and 3 grandchildren. He is survived by his loving partner, Carol Monroe; and by two children from his first marriage, a son, Thomas Meissner; a daughter, Suzanne Jacobs, and four grandchildren. 

Born in Berlin, Germany, September 22, 1941, he was the son of the late Hellmuth Meissner and Edith Maria Strauch Meissner. At age 11, he migrated with his parents to Guelph, Ontario, then in 1956 to the United States. Prior to moving to Nelson Country in 1997, he lived in New Jerseyand Arizona. He was a graduate of the Pennington School for Boys (Pennington, NJ), and attended Phoenix College, (Phoenix, AZ), andArizona State University. 

Jim accumulated all the tools and electrical gear he could. He put together a workshop in the basement of the family home when he was in his early teens, and added to it all his life. At age 16, he won first place in the New Jersey State Science Fair for designing and building a complete hi-fi system. 

Jim always loved music. He acted and sang in professional theater, sang in the Bach Madrigal Society and the Phoenix Symphonic Choir, sang in the church choir and was in several musical plays. He was a member of the touring choirs of Phoenix College and Arizona State University. In 1960, he won the seldom-awarded Trudel Award for excellence in music. 

While attending college, he earned money by repairing radios, televisions, and phonographs and building hi-fi systems and enclosures. He opened a recording studio, designing and building all the necessary electronic equipment. He later designed, patented and built stereo speakers that worked on a new principle that resulted in uniform sound regardless of placement. 

After college, he was for many years employed as a consultant for companies in the aerospace, electronics and health-care industries. In addition to his consulting business, he purchased and for ten years managed a company that made transistors and capacitors. Among the companies he consulted for were Goodyear Aerospace Corp., North American Aviation, Arizona Electronics Standards Lab, Julie Research Laboratories,  RFL Industries, and many more. 

Jim’s unique understanding of how things worked, his incessant curiosity, and his skill in researching and experimenting led him to produce inventions in the fields of energy, health, and food production. Always interested in enhancing human performance, he developed ways to retrain the brain using electronics. One such device, which he called the Brain State Synchronizer, could be used to improve performance in sports and other activities.  He was the holder of numerous patents. 

Jim’s greatest joy in life was helping people. Perhaps the most important project of his later years was sharing the knowledge he had acquired through research into the causes and treatment of cancer, Lyme disease, and other ailments through personal contact and a health forum he ran on the internet. Since his own illness became known, he received hundreds of encouraging and grateful messages from people from all over the country, saying how much he had helped them. 

Condolences can be sent to http://meissnerresearch.com/contact/.  A celebration of his life will be held in mid-January. 

The family requests that memorial donations be made to the Hospice of thePiedmont, 675 Peter Jefferson Parkway, Suite 300, Charlottesville, VA22911, in appreciation of their support through the last months of his illness.

Holy Cow, It Works! New rife testimonial

This was posted on one of the Lyme/Rife groups recently, I thought I would share:

About three years ago, the balls of my feet began to hurt and my toes were going numb while I was running. It got worse and worse until it was hard to walk too many steps in a day. 3 podiatrists, cortisone shots, sonograms, and an MRI turned up nothing. 

My wife of 18+ years has had Lyme for 25+ years, but was just diagnosed a few months ago. Working on a guess that I might have contracted Lyme from her (as an STD), I bought a used EMEM from a nice person on this board.

I gave myself a treatment directed at my feet (2m@432, 2m@800, 2m@4328, 2m@10K) before bed one night last week. I could feel electrical pops every couple of seconds in my feet at 432 and 800. I woke up the next morning with no pain in my feet. It lasted for five days, longer than I’ve been pain free for a couple of years. I’ve followed it up with another treatment to not as good an effect, but I’m patient.

In a word, I’m stunned. I was a HUGE skeptic, and here I have evidence it works, at least for me.

I don’t know that I have lyme, but I apparently have something, and the EMEM is working.

How old is Lyme disease? National Geographic: 5,000 years old

Maybe Lyme disease was around a long time before Plum Island. That is the position of National Geographic:


This is a fascinating story; perhaps Lyme-related infections have been pestering humanity for thousands of years, and may even be responsible for many of the health problems of our ancestors.

$5 off at iHerb.com, my favorite supplement store

I love iHerb.com. It reminds me a lot of Amazon.com – cheap prices, reliable shipping / service, great customer reviews for supplement products, huge selection, free shipping over $20. I encourage you to try it for your next supplement order.

Enter coupon code HES111 and you will receive $5 off your first order. Visit iHerb.com today.


Lyme Disease Symptom Checklist


James Schaller, M.D., M.A.R.

The following checklist is not meant to be complete or authoritative. Information about Lyme disease is constantly emerging and changing. Therefore any checklist is intended for use as a starting point. In traditional medicine, a physician performs a complete history and physical. Labs and studies assist in clarifying the differential diagnosis. In Lyme disease, much debate exists about laboratory kits, the alteration of kits to have fewer possible bands, and which labs are optimally sensitive and specific. This checklist is not intended to address that issue or treatment.

Over 200 vectors carry the Ixodes tick, which is the most commonly known insect spreading Lyme disease. With so many vectors, the underlying assumption behind this checklist is that Lyme is not rare in North America, Europe, South America, Russia, Africa or Asia.

We know Lyme disease is highly under-reported.

Immediately upon the onset of a tick bite, it transmits a pain killer, anti-histamine and an anti-coagulant. Based on animal studies, it is also possible the bulls-eye rash is less common then assumed, in part because injections of spirochete related material in laboratory animals only show a rash with the second injection.

This checklist is offered with the sincere wish that others will improve on it. It is this author’s personal belief that tick and flea-borne infection medicine is as specialized as HIV and Hepatitis medical science.

Some of the checklist materials might be new to you, which underscores the need for another scale to add to the ones currently in existence. This list is based on a massive review of thousands of papers over a decade of full-time reading, 2012 science revelations, and/or massive chart reviews. Since modern Lyme disease seems to focus on tick borne disease and other laboratory testing, I will start with lab testing considerations. If a lab test has a value or a percentage, the numbers I am picking are meant to avoid missing positive patients. I am concerned about physicians and other healthcare workers not treating an infected patient, who over time can experience disability or death at a frequency that is impossible to determine.


1. Vitamin D level is in the lowest 20%. If you supplement, it should be in top 50%.
2. CD57 or CD58 is in the lowest 20th percentile
3. Free testosterone is in 10th percentile or below
4. In 5% of patients the testosterone or free testosterone is over the normal range.
5. DHEA is in lower 20%. Or rarely is it fully over the top level.
6. Free dihydrotestosterone is in the lowest 20th percentile or well over the normal range.
7. Epstein Barr Virus is abnormal in any measure. [This virus is believed to be positive over normal positive levels in the presence of infections or high inflammation.]
8. On the Western Blot, IgG or IgM any species specific band at any blood level, e.g., 18, 21, 23, 30, 31, 34, 37, 39, 83, 93.
9. A free T3 level under 2.8 [the normal bottom range in 1990 was 2.6; the influx of large numbers of elderly patients reset the healthy “normal” range].
10. Positive for viruses such as CMV, HHP-6, Coxsackie B Types 1, 2, 3, 4, 5, 6, Parvo B-19 or Powassan virus
11. Positive for Mycoplasma, e.g. mycoplasma pneumoniae.
12. The patient is positive for infections other than routine Lyme, [that is Borrelia burgdorferi sensu stricto, Borrelia afzelii and Borrelia garinii]. Some of the other infections also carried by infectious ticks, fleas or other vectors include Babesia (duncani, microti or other), Anaplasma (HGA), Ehrlichia (various species/strains), Rocky Mountain or other Spotted Fevers, Brucellosis, Leptospirosis, Q-fever, STARI (Master’s Disease), Malaria, and Bartonella [e.g., B. henselae, B. quintana, B. elizabethae and B. melophagi]. Once tests are commercially available for testing all forms of protozoa affecting humans, including FL1953, all Bartonella species, and Borrelia miyamotoi and other Lyme species, reporting should increase.
13. IL-B is in lowest 10th percentile
14. IL-6 is in lowest 10th percentile
15. TNF-alpha is under 2, or in lowest 20th percentile
16. A WBC count was, or is, under 4.5
17. Eosinophil level in the CBC manual exam is either at 0-1 or 6-7
18. Total manual Eosinophil level is 140 or less
19. XRAY or other study shows cartilage defects in excess of injury or age median
20. If a full auto-immunity panel is run with at least eight different tests, two are positive; for example, you have a positive anti-gliadin and a positive thyroid peroxidase.
21. Positive or near positive (borderline) ELISA, PCR, or a positive tissue biopsy; or a tick from your body is positive for Lyme or other tick infection
22. Lab tests show high inflammation, e.g., a high C4a, elevated cholesterol and C-peptide. These are never specific just for Lyme
23. Lab tests show a MSH level under 30 [the reference range of 0-40 is due to the increase of very sick patients tested, and 40-85 is a better reference range which was used before the flood of the sick reset the range of normal]. MSH is an anti-inflammatory hormone.
24. VIP is under 20. This is an anti-inflammation chemical.


25. Weight loss or gain in excess of 20 pounds in 12 weeks
26. A round or oval rash with a dark center was or is present in a loose “bulls-eye pattern.” Other size and shape rashes that have no other cause after exposure to ticks and vectors.
27. Healing is slow after scratches or surgery. For example, after a cat scratch, flea bite or tick bite the mark is still visible later.
28. Skin on arms, hands or feet has a texture like rice paper.
29. Clear reaction and effect seen with antibiotic treatment. Specifically, a marked improvement or worsening of a serious medical problem or function is observed with a spirochete killing treatment, e.g., doxycycline, tetracycline, minocycline, any penicillin such as amoxicillin, azithromycin, clarithromycin or cefuroxime.
30. Presence of skin tags, red papules of any size, excess blood vessels compared to peers, and stretch marks with color or in significant excess of peers.
31. Moles and raised or hard plaques in excess of the few on normal skin.
32. Areas of skin with ulcerations such as those seen in syphilis, but at any location on the body.
33. Areas of clear hypo-pigmentation and hyper-pigmentation
34. Positive ACA (Acrodermatitis chronica atrophicans) which is a sign of long term untreated Lyme disease. Some report ACA begins as a reddish-blue patch of discolored skin, often of the hands or feet. It may include the back in some patients. The lesion slowly atrophies over months to years, with many developing skin that is thin, dry, hairless, wrinkled and abnormally colored. The color of the extremities such as hands and feet can be red, dark red, brown, dark blue or purple.
35. Patient’s short-term memory is poor. For example, if asked to recall these numbers—23, 5, 76, 43 and 68—the patient cannot recall them.
36. Patient cannot reverse four numbers, so if given—18, 96, 23 and 79—the patient cannot do it.
37. If asked to subtract 17 from 120, (college graduate), it cannot be done in a timely manner. If a high school graduate, subtract 7 from 100 and continue to subtract by 7 four times in 20 seconds.
38. Light headedness upon standing quickly in excess of peers, and with no clear cause
39. Dizziness unrelated to position
40. Dizziness made worse by Lyme killing antibiotics
41. Trouble doing a nine step heel to toe straight line walk test with fingers slightly in pockets [The patient should not sway or need their hands pulled out to prevent a fall]. In patients with past experience in skating, skiing, dance or ballet this should be very easy and is rarely a challenge to such people. If it is not easy, it is suspicious medically, but not only for Lyme disease.
42. Trouble performing a one leg lift, in which one leg is lifted 12-18 inches off the ground in front of you, as you count, e.g., “one Mississippi, two Mississippi, etc.”
43. Positive nystagmus [your eye jerks when you look right or left]


44. Illnesses that come and go and decrease functioning with no certain cause
45. Serious illnesses that undermine function with no clear cause, and which affect more than one body organ
46. An abnormal lab result, physical exam finding or illness that is given many diagnoses or has no clear cause.
47. Mild to severe neurological disorders or psychiatric disorders
48. A very profound neurological disease which does not clearly fit the labs, studies and course of the illness
49. A moderate or severe medical, psychiatric or neurological illness. [Many severe disorders can be associated with spirochetes such as those causing syphilis, and some propose that Lyme is also related to a well-known serious brain disease].
50. Severe medical, psychiatric or neurology illness with uncommon features, such as Parkinson’s disease, appearing at a young age
51. Facial paralysis (Bell’s palsy)
52. Personality has changed negatively and significantly for no clear reason.
53. Psychosis at any age, but especially after 40 years of age when usually it would have already manifested itself
54. Severe anxiety
55. Mania or profound rage
56. Depression
57. Depression or anxiety that did not exist when you were less than 25 years of age
58. Irritability
59. Any one of the following: paranoia, dementia, schizophrenia, bipolar disorder, panic attacks, major depression, anorexia nervosa or obsessive-compulsive disorder.
60. Adult onset ADHD/ADD [Primary psychiatric biological ADD or ADHD is present at 7 years of age. Adult onset is a sign of a medical condition.]
61. Increased verbal or physical fighting with others
62. Functioning at work or in parenting is at least 20% reduced
63. Patience and relational skills are decreased by 20% or more
64. A mild to profound decrease of insight, i.e., an infected patient does not see their decreased function, failed treatment or personality change
65. A new eccentric rigidity to hearing new medical or other important information
66. Difficulty thinking or concentrating
67. Poor memory and reduced ability to concentrate
68. Increasingly difficult to recall names of people or things
69. Difficulty speaking or reading
70. Difficulty finding the words to express what you want to say
71. Inability to learn new information as well as in the past [receptive learning]
72. Repeating stories or forgetting information told to close relations, such as a spouse, roommate, sibling, best friend or parent
73. Confusion without a clear reason
74. An addiction that results in relapse in spite of sincere, reasonable and serious efforts to stop
75. Fatigue in excess of normal, or fatigue that is getting worse
76. Trouble sleeping including mild to severe insomnia and disrupted sleep
77. Sleep in excess of 9 hours a day or night, or sleeping in excess of 9 hours every day if allowed
78. Trouble falling asleep
79. Trouble staying asleep [Taking a 5 minute bathroom break does not count]
80. Gastritis or stomach sensitivity not caused by H. Pylori
81. Intestinal troubles that are unable to be fully managed and/or which have no clear diagnosis
82. Nausea without a clear reason
83. Sensitivity to lights, sounds, touch, smell or unusual tastes
84. Sensitivity to cleaning chemicals, fragrances and perfumes
85. Ear problems such as pain or increased ear “pressure.”
86. Any trouble with the senses (vision, sound, touch, taste or smell). The use of corrective lenses or contacts does not count, unless the prescription is changed more than expected.
87. Buzzing or ringing in ears
88. Double vision, floaters, dry eyes, or other vision trouble
89. Conjunctivitis (pinkeye) or occasional damage to deep tissue in the eyes
90. Blood clots fast when you get a cut, or you have a diagnosed problem with clotting. This may also be seen in blood draws where blood draw needle clots when blood is being removed. If on a blood thinner, blood thinness level goes up and down too much.
91. Cardiac impairment
92. Chest pain with all labs and studies in normal range
93. Occasional rapid heartbeats (palpitations)
94. Heart block/heart murmur
95. Heart valve prolapse
96. Shortness of breath with no clear cause on pulmonary function tests, examination, lab testing, X-rays, MRI’s, etc.
97. Air hunger or feelings of shortness of breath
98. Someone in your neighborhood within 400 yards in any direction of your dwelling has been diagnosed with a tick borne infection. [This includes vacation locations].
99. You have someone living with you with any type of tick-borne infection—this assumes they were not merely tested for one infection. [It is not proven that the small Lyme-carrying ticks only carry Lyme, and it is possible some carry other infections without carrying Lyme at all].
100. You have removed any ticks from your body in your lifetime.
101. You have removed ticks from your clothing in your lifetime.
102. After a tick or bug bite, you had a fever for at least 48 hours.
103. After a tick or bug bite, you were ill.
104. Grew up or played in areas with many small wild mammals.
105. When you are in a room that has visible mold or smells like mold and you start to feel ill, you do not return to your baseline health in 24 hours.
106. Any discomfort within two minutes of being in a musty or moldy location
107. Gaining or losing weight in a manner clearly inconsistent with diet and exercise
108. New or more food allergies than ten years ago
109. Feel worse after eating breads, pasta or sweets
110. No longer tolerate or enjoy alcohol
111. Anti-histamines are bothersome, more so than in the past.
112. Reaction to medications is excessive (you are very “sensitive” to medications)
113. Your response to antibiotics is significantly positive and you feel more functional, or you have the opposite reaction and feel worse, feeling ill, fatigued or agitated.
114. Numbness, tingling, burning, or shock sensations in an area of skin
115. One or more troublesome skin sensations that move over months or years and do not always stay in one location
116. Rash or rashes without a simple and obvious cause
117. Rashes that persist despite treatment
118. Eccentric itching with no clear cause
119. Hair loss with no clear cause
120. Muscle pain or cramps
121. Muscle spasms
122. Muscle wasting without a clear cause
123. Trouble with your jaw muscle(s) or joint insomnia (TMJ)
124. Joint defects in one joint with no clear cause if 20 or younger
Joint defects in two joints or more if 35 or younger
Joint defects in three or more locations if younger than 55 with no clear trauma
125. Swelling or pain (inflammation) in the joints. [Most patients never have joint disease].
126. Joint pain that shifts location
127. Neck stiffness
128. Chronic arthritis with or without episodes of swelling, redness, and fluid buildup
129. Chronic pain in excess of what seems reasonable
130. Nerve pain without a clear cause
131. Headaches that do not respond fully to treatment, or which are getting worse
132. New allergies or increased allergies over those of your peers
133. Any autoimmunity–Lyme and other tick infections, over many years, increase inflammation and decrease anti-inflammation chemicals. We believe this leads to increased food sensitivities, increased autoimmunity and a heightened sensitivity to various chemicals and medications.
134. Day time sweats
135. Night time sweats
136. Chills
137. Flu-like symptoms
138. Bladder dysfunction of any kind
139. Treatment resistant interstitial cystitis
140. Abnormal menstrual cycle
141. Decreased or increased libido
142. Increased motion sickness
143. Fainting
144. A spinning sensation or vertigo


145. Pets or farm animals positive with ANY tick borne virus, bacteria or protozoa, or clinical symptoms without a clear diagnosis or cause.
146. The patient’s mother is suspected of having or has been diagnosed with Babesia, Ehrlichia, Rocky Mountain Spotted Fever, Anaplasma, Lyme, Bartonella or other tick borne disease based on newer direct and indirect testing, or clinical signs and symptoms.
147. A sibling, father, spouse or child with any tick borne infection
148. Casual or work-related exposure to outdoor environments with brush, wild grasses, wild streams or woods (Examples- golf courses, parks, gardens, river banks, swamps, etc.)
149. Pets, e.g., horses, dogs or cats, have had outdoor exposures to areas such as brush, wild grasses, wild streams or woods.
150. Exposure to ticks in your past homes
151. Clear exposure to ticks during vacations or other travels
152. You played in grass in the past.
153. You have been bitten by fleas.
154. You have been scratched by a cat or dog.


Some of the above listed signs and symptoms fit other infections that may be more common than Lyme disease. Unfortunately, the research and experience indicating diverse infections carried by the Ixodes and other ticks is ignored. Further, “testing” usually involves one test for a mono-infection–Borrelia or Lyme. Ticks and other vectors should never be assumed to carry only Lyme disease.

Please note that when we are talking about the Ixodes tick we are not referring to this as a “deer tick” since it has over 200 vectors (Ostfeld). Many of the tick reduction options presently suggested are not successful in accomplishing their goals. Reducing deer populations, once thought to reduce tick populations and incidence of Lyme disease, may simply increase tick numbers in mammals and other carriers that live closer to humans.

All healers have their familiar way of thinking, testing and treating. Kuhn has shown we are all biased and struggle to be objective. Further, tick and flea infections have almost infinite pathological effects because the human body and these clusters of infections are so complex. I have not suggested a grid or a set number of symptoms, because one would not fit this list. Simply, the goal of this checklist is to have you think broadly.
You cannot use this checklist to diagnose Lyme disease or to rule it out.
A Lyme checklist is very medically important, since it is still an emerging illness and can sometimes disable or increase mortality risk in patients of any age if not diagnosed and treated early in the infection.
Writings in the past fifteen years have either viewed Babesia and Bartonella as mere “co-infections,” or a footnote of a spirochetal infection [i.e., Lyme]. Either infection can hide for decades, and then possibly disable or kill a person by causing a clot, heart arrhythmia or by other means.
The detection of Lyme from stained tissue samples or blood is very difficult. Currently, the well-established indirect lab test patterns presented are not used or understood by all health care professionals. While this is fully understandable, I hope it may change in the coming decade. Tick infections have systemic impacts on the body, and are not limited to effects reported in journal articles, a few books or any national or international guidelines.
Dr. Schaller has published the four most recent textbooks on Babesia and the only recent textbook in any language on Bartonella. His most recent book on Lyme, Babesia and Bartonella includes a “researchers only” list of over 2,600 references considered to be a start for basic education in tick infection medicine.
He published articles on both Babesia as a cancer primer and Bartonella as a profound psychiatric disease under the supervision of the former editor of the Journal of the American Medical Association (JAMA). He also published entries on multiple tick and flea-borne infections, including Babesia, Bartonella and Lyme disease, in a respected infection textbook endorsed by the NIH Director of Infectious Disease.
Dr. Schaller is the author of seven texts on tick and flea-borne infections. He is rated a TOP and BEST physician, with the latter being awarded to only 1 in 20 physicians by physician ratings. He is also rated a TOP physician by patients, again ranking in the top 1 in 20 physicians.
This form may not be altered if it is printed or posted, in any manner, without written permission. It can be printed for free to assist in diagnostic reflections, as long as no line is redacted or altered, including the introduction or final paragraphs. Dr. Schaller does not claim that this is a flawless or final form, and defers all diagnostic decisions to your licensed health professional.


Aalto A, Sjöwall , Davidsson L, Forsberg P, Smedby O. Brain magnetic resonance imaging does not contribute to the diagnosis of chronic neuroborreliosis. Acta Radiol. 2007 Sep;48(7):755-62. PMID:17729007

Aberer E. [Neuroborreliosis or Borrelia hysteria. This case becomes a nightmare!].[Article in German]. MMW Fortschr Med. 2006 Nov 9;148(45):8. PMID:17615738

Aboul-Enein F, Kristoferitsch W. Normal pressure hydrocephalus or neuroborreliosis? Wien Med Wochenschr. 2009;159(1-2):58-61. PMID:19225737

Alaedini A, Latov N. Antibodies against OspA epitopes of Borrelia burgdorferi cross-react with neural tissue. J Neuroimmunol. 2005 Feb;159(1-2):192-5. Epub 2004 Nov 26. PMID:15652419

Angelakis E, Billeter SA, Breitschwerdt EB, Chomel BB, Raoult D. Potential for tick-borne bartonellosis. Emerg Infect Dis. 2010 Mar;16(3):385-91.

Auwaerter PG. Point: antibiotic therapy is not the answer for patients with persisting symptoms attributable to lyme disease. Clin Infect Dis. 2007 Jul 15;45(2):143-8. Epub 2007 Jun 5. PMID:17578771

Banarer M, Cost K, Rychwalski P, Bryant KA. Chronic lymphocytic meningitis in an adolescent. J Pediatr. 2005 Nov;147(5):686-90. PMID:16291364

Baneth G, Breitschwerdt EB, Hegarty BC, Pappalardo B, Ryan J. A survey of tick-borne bacteria and protozoa in naturally exposed dogs from Israel. Vet Parasitol. 1998 Jan 31;74(2-4):133-42.

Barbour AG. Laboratory aspects of Lyme borreliosis. Clin Microbiol Rev 1988 Oct;1(4):415-31.

Barie PS. Warning! Danger Will Robinson! Lyme disease clinical practice guidelines of the Infectious Diseases Society of America, activist patients, antitrust law, and prosecutorial zeal. Surg Infect (Larchmt). 2007 Apr;8(2):147-50. PMID:17437359

Batinac T, Petranovic D, Zamolo G, Petranovic D, Ruzic A. Lyme borreliosis and multiple sclerosis are associated with primary effusion lymphoma. Med Hypotheses. 2007;69(1):117-9. Epub 2007 Jan 2. PMID:17197115

Begon E. [Lyme arthritis, Lyme carditis and other presentations potentially associated to Lyme disease].[Article in French]. Med Mal Infect. 2007 Jul-Aug;37(7-8):422-34. Epub 2007 Aug 14. PMID:17698309

Benhnia MR, Wroblewski D, Akhtar MN, Patel RA, Lavezzi W, Gangloff SC, Goyert SM, Dvoráková J, Celer V. [Pharmacological aspects of Lyme borreliosis].[Article in Czech]. Ceska Slov Farm. 2004 Jul;53(4):159-64. PMID:15369225

Bhate C, Schwartz RA. Lyme disease: Part II. Management and prevention. J Am Acad Dermatol. 2011 Apr;64(4):639-53; quiz 654, 653. PMID:21414494

Biesiada G, Czapiel J, Sobczyk-Krupiarz I, Garlicki A, Mach T. Neuroborreliosis with extrapyramidal symptoms: a case report. Pol Arch Med Wewn. 2008 May;118(5):314-7. PMID:18619183

Billeter SA, Levy MG, Chomel BB, Breitschwerdt EB. Vector transmission of Bartonella species with emphasis on the potential for tick transmission. Med Vet Entomol. 2008 Mar;22(1):1-15.
Bitar I, Lally EV. Musculoskeletal manifestations of Lyme disease. Med Health R I. 2008 Jul;91(7):213-5. PMID:18705221

Blanc F. [Epidemiology of Lyme borreliosis and neuroborreliosis in France].[Article in French]. Rev Neurol (Paris). 2009 Aug-Sep;165(8-9):694-701. Epub 2009 May 17. PMID:19447458

Blanc F; GEBLY. [Neurologic and psychiatric manifestations of Lyme disease].[Article in French]. Med Mal Infect. 2007 Jul-Aug;37(7-8):435-45. Epub 2007 Mar 9. PMID:17350199

Bransfield RC, Wulfman JS, Harvey WT, Usman AI. The association between tick-borne infections, Lyme borreliosis and autism spectrum disorders. Med Hypotheses. 2008;70(5):967-74. Epub 2007 Nov 5. PMID:17980971

Brehm M, Rellecke P, Strauer BE. [Inflammatory cardiac diseases by primary extracardial diseases].[Article in German]. Internist (Berl). 2008 Jan;49(1):27-33. PMID:17992497

Breitschwerdt EB. Feline bartonellosis and cat scratch disease. Vet Immunol Immunopathol. 2008 May 15;123(1-2):167-71. Epub 2008 Jan 19. Review.

Breitschwerdt EB, Atkins CE, Brown TT, Kordick DL, Snyder PS. Bartonella vinsonii subsp. berkhoffii and related members of the alpha subdivision of the Proteobacteria in dogs with cardiac arrhythmias, endocarditis, or myocarditis. J Clin Microbiol. 1999 Nov;37(11):3618-26.

Breitschwerdt EB, Blann KR, Stebbins ME, Muñana KR, Davidson MG, Jackson HA, Willard MD. Clinicopathological abnormalities and treatment response in 24 dogs seroreactive to Bartonella vinsonii (berkhoffii) antigens. J Am Anim Hosp Assoc. 2004 Mar-Apr;40(2):92-101.

Breitschwerdt EB, Hegarty BC, Hancock SI. Sequential evaluation of dogs naturally infected with Ehrlichia canis, Ehrlichia chaffeensis, Ehrlichia equi, Ehrlichia ewingii, or Bartonella vinsonii. J Clin Microbiol. 1998 Sep;36(9):2645-51.

Breitschwerdt EB, Hegarty BC, Maggi R, Hawkins E, Dyer P. Bartonella species as a potential cause of epistaxis in dogs. J Clin Microbiol. 2005 May;43(5):2529-33.

Breitschwerdt EB, Kordick DL. Bartonellosis. J Am Vet Med Assoc. 1995 Jun 15;206(12):1928-31. Review.

Breitschwerdt EB, Kordick DL. Bartonella infection in animals: carriership, reservoir potential, pathogenicity, and zoonotic potential for human infection. Clin Microbiol Rev. 2000 Jul;13(3):428-38. Review.

Breitschwerdt EB, Kordick DL, Malarkey DE, Keene B, Hadfield TL, Wilson K. Endocarditis in a dog due to infection with a novel Bartonella subspecies. J Clin Microbiol. 1995 Jan;33(1):154-60.

Breitschwerdt EB, Maggi RG. A confusing case of canine vector-borne disease: clinical signs and progression in a dog co-infected with Ehrlichia canis and Bartonella vinsonii ssp. berkhoffii. Parasit Vectors. 2009 Mar 26;2 Suppl 1:S3.

Breitschwerdt EB, Maggi RG. Comparative medical features of canine and human bartonellosis. Clin Microbiol Infect. 2009 Dec;15 Suppl 2:106-7. Epub 2009 Apr 30.

Breitschwerdt EB, Maggi RG, Cadenas MB, de Paiva Diniz PP. A groundhog, a novel Bartonella sequence, and my father’s death. Emerg Infect Dis. 2009 Dec;15(12):2080-6.

Breitschwerdt EB, Maggi RG, Chomel BB, Lappin MR. Bartonellosis: an emerging infectious disease of zoonotic importance to animals and human beings. J Vet Emerg Crit Care (San Antonio). 2010 Feb;20(1):8-30. Review.

Breitschwerdt EB, Maggi RG, Duncan AW, Nicholson WL, Hegarty BC, Woods CW. Bartonella species in blood of immunocompetent persons with animal and arthropod contact. Emerg Infect Dis. 2007 Jun;13(6):938-41.

Breitschwerdt EB, Maggi RG, Farmer P, Mascarelli PE. Molecular evidence of perinatal transmission of Bartonella vinsonii subsp. berkhoffii and Bartonella henselae to a child. J Clin Microbiol. 2010 Jun;48(6):2289-93. Epub 2010 Apr 14.

Breitschwerdt EB, Maggi RG, Lantos PM, Woods CW, Hegarty BC, Bradley JM. Bartonella vinsonii subsp. berkhoffii and Bartonella henselae bacteremia in a father and daughter with neurological disease. Parasit Vectors. 2010 Apr 8;3(1):29.

Breitschwerdt EB, Maggi RG, Nicholson WL, Cherry NA, Woods CW. Bartonella sp. bacteremia in patients with neurological and neurocognitive dysfunction. J Clin Microbiol. 2008 Sep;46(9):2856-61. Epub 2008 Jul 16.

Breitschwerdt EB, Maggi RG, Robert Mozayeni B, Hegarty BC, Bradley JM, Mascarelli PE. PCR amplification of Bartonella koehlerae from human blood and enrichment blood cultures. Parasit Vectors. 2010 Aug 24;3:76.

Breitschwerdt EB, Maggi RG, Sigmon B, Nicholson WL. Isolation of Bartonella quintana from a woman and a cat following putative bite transmission. J Clin Microbiol. 2007 Jan;45(1):270-2. Epub 2006 Nov 8.

Breitschwerdt EB, Maggi RG, Varanat M, Linder KE, Weinberg G. Isolation of Bartonella vinsonii subsp. berkhoffii genotype II from a boy with epithelioid hemangioendothelioma and a dog with hemangiopericytoma. J Clin Microbiol. 2009 Jun;47(6):1957-60. Epub 2009 Apr 15.

Breitschwerdt EB, Mascarelli PE, Schweickert LA, Maggi RG, Hegarty BC, Bradley JM, Woods CW. Hallucinations, sensory neuropathy, and peripheral visual deficits in a young woman infected with Bartonella koehlerae. J Clin Microbiol. 2011 Sep;49(9):3415-7. Epub 2011 Jul 6.

Breitschwerdt EB, Sontakke S, Cannedy A, Hancock SI, Bradley JM. Infection with Bartonella weissii and detection of Nanobacterium antigens in a North Carolina beef herd. J Clin Microbiol. 2001 Mar;39(3):879-82.

Breitschwerdt EB, Suksawat J, Chomel B, Hegarty BC. The immunologic response of dogs to Bartonella vinsonii subspecies berkhoffii antigens: as assessed by Western immunoblot analysis. J Vet Diagn Invest. 2003 Jul;15(4):349-54.

Brtkova J, Jirickova P, Kapla J, Dedic K,, Pliskova L. Borrelia arthritis and chronic myositis accompanied by typical chronic dermatitis. JBR-BTR. 2008 May-Jun;91(3):88-9. PMID:18661710

Burns RB, Hartman EE. A 58-year-old man with a diagnosis of chronic Lyme disease, 1 year later. JAMA. 2003 Dec 24;290(24):3247. PMID:14693878

Caimano MJ, Radolf JD, Sellati TJ. Signaling through CD14 attenuates the inflammatory response to Borrelia burgdorferi, the agent of Lyme disease. J Immunol. 2005 Feb 1;174(3):1539-48. PMID:15661914

Calza L, Manfredi R, Chiodo F. [Tick-borne infections].[Article in Italian]. Recenti Prog Med. 2004 Sep;95(9):403-13. PMID:15473378

Cameron D. Obstacles to trials of chronic Lyme disease in actual practice. Minerva Med. 2009 Oct;100(5):435-6. PMID:19910896

Cameron DJ. Clinical trials validate the severity of persistent Lyme disease symptoms. Med Hypotheses. 2009 Feb;72(2):153-6. Epub 2008 Nov 13. PMID:19013025

Cameron DJ. Proof that chronic lyme disease exists. Interdiscip Perspect Infect Dis. 2010;2010:876450. Epub 2010 May 25. PMID:20508824

Cerar T, Ruzic-Sabljic E, Cimperman J, Strle F. Comparison of immunofluorescence assay (IFA) and LIAISON in patients with different clinical manifestations of Lyme borreliosis. Wien Klin Wochenschr. 2006 Nov;118(21-22):686-90. PMID:17160608

Chandra A, Wormser GP, Klempner MS, Trevino RP, Crow MK, Latov N, Alaedini A. Anti-neural antibody reactivity in patients with a history of Lyme borreliosis and persistent symptoms. Brain Behav Immun. 2010 Aug;24(6):1018-24. Epub 2010 Mar 18th PMID:20227484

Chernogor LI, Arbatskaia EV, Danchinova GA, Kozlova IV, Gorina MO, Suntsova OV, Chaporgina EA, Belikov SI, Borisov VA. [Clinical and laboratory characterization of Ixodes tick-borne borreliosis in the Baikal area].[Article in Russian]. Zh Mikrobiol Epidemiol Immunobiol. 2005 Nov-Dec;(6):60-2. PMID:16438378

Chomel BB, Boulouis HJ, Maruyama S, Breitschwerdt EB. Bartonella spp. in pets and effect on human health. Emerg Infect Dis. 2006 Mar;12(3):389-94. PMID 16704774
Clarissou J, Song A, Bernedo C, Guillemot D, Dinh A, Ader F, Perronne C, Salomon J. Efficacy of a long-term antibiotic treatment in patients with a chronic Tick Associated Poly-organic Syndrome (TAPOS). Med Mal Infect. 2009 Feb;39(2):108-15. Epub 2009 Jan 4. PMID:19124209

Comer JA, Diaz T, Vlahov D, Monterroso E, Childs JE. Evidence of rodent-associated Bartonella and Rickettsia infections among intravenous drug users from Central and East Harlem, New York City. Am J Trop Med Hyg. 2001 Dec;65(6):855-60. PMID:11791987

Comer JA, Flynn C, Regnery RL, Vlahov D, Childs JE. Antibodies to Bartonella species in inner-city intravenous drug users in Baltimore, Md. Arch Intern Med. 1996 Nov 25;156(21):2491-5. PMID:8944742

Coyle PK. Lyme disease. In: Feldmann E, ed. Current diagnosis in neurology. St Louis:Mosby,1994; pp 110-4.

Coyle PK ed. Lyme Disease. St. Louis:Mosby Year Book 1993; pp 187-91.

Clark JR, Carlson RD, Sasaki CT, Pachner AR, Steere AC. Facial paralysis in Lyme disease. Laryngoscope 1985 Nov;95(11):1341-5.

Créange A. [Clinical manifestations and epidemiological aspects leading to a diagnosis of Lyme borreliosis: neurological and psychiatric manifestations in the course of Lyme borreliosis].[Article in French]. Med Mal Infect. 2007 Jul-Aug;37(7-8):532-9. Epub 2007 Mar 26. PMID:17368785

da Franca I, Santos L, Mesquita T, Collares-Pereira M, Baptista S, Vieira L, Viana I, Vale E, Prates C. Lyme borreliosis in Portugal caused by Borrelia lusitaniae? Clinical report on the first patient with a positive skin isolate. Wien Klin Wochenschr. 2005 Jun;117(11-12):429-32. PMID:16053200

Danz B, Kreft B, Radant K, Marsch WCh, Fiedler E. Skin-coloured facial oedema as an initial manifestation of acrodermatitis chronica atrophicans. J Eur Acad Dermatol Venereol. 2008 Jun;22(6):751-3. PMID:18482035

Dattwyler RJ, Halperin JJ, Volkman DJ, Luft BJ. Treatment of late Lyme borreliosis – randomized comparison of ceftriaxone and penicillin. Lancet 1988 May 28;1(8596):1191-4.

Dattwyler RJ, Luft BJ, Maladorno D, et al. Treatment of late Lyme disease – a comparison of 2 weeks vs 4 weeks of ceftriaxone. VII International Congress on Lyme Borreliosis. San Francisco, June, 1996.

Dattwyler RJ, Wormser GP, Rush TJ, Finkel MF, Schoen RT, Grunwaldt E, Franklin M, Hilton E, Bryant GL, Agger WA, Maladorno D. A comparison of two treatment regimens of ceftriaxone in late Lyme disease. Wien Klin Wochenschr. 2005 Jun;117(11-12):393-7. PMID:16053194

de Freitas MR. Infectious neuropathy. Curr Opin Neurol. 2007 Oct;20(5):548-52. PMID:17885443

De Heller-Milev M, Peter O, Panizzon RG, Laffitte E. [Borrelial erythema of the face].[Article in French]. Ann Dermatol Venereol. 2008 Dec;135(12):852-4. Epub 2008 Oct 26. PMID:19084697

DeLong A. Lyme disease. Med Health R I. 2008 Dec;91(12):390; author reply 390. PMID:19170319

DePietropaolo DL, Powers JH, Gill JM, Foy AJ. Diagnosis of Lyme disease. Del Med J. 2006 Jan;78(1):11-8. PMID:16548394

Dillon R, O’Connell S, Wright S. Lyme disease in the U.K.: clinical and laboratory features and response to treatment. Clin Med. 2010 Oct;10(5):454-7. PMID:21117376

Djukic M, Schmidt-Samoa C, Nau R, von Steinbüchel N, Eiffert H, Schmidt H. The diagnostic spectrum in patients with suspected chronic Lyme neuroborreliosis–the experience from one year of a university hospital’s Lyme neuroborreliosis outpatients clinic. Eur J Neurol. 2011 Apr;18(4):547-55. Epub 2010 Oct 27. PMID:20977545

Drancourt M, Tran-Hung L, Courtin J, Lumley H, Raoult D. Bartonella quintana in a 4000-year-old human tooth. J Infect Dis. 2005 Feb 15;191(4):607-11.

Dressler F, Whalen JA, Reinhardt BN, Steere A. Western blotting in the serodiagnosis of Lyme disease. J Infect Dis 1993 Feb;167(2):392-400.

Egle UT. [Chronic borreliosis? No, psychosomatic illness! (interview by Dr. med. Brigitte Moreano)].[Article in German]. MMW Fortschr Med. 2005 May 26;147(21):15. PMID:15966166

Einecke U. [Winter pause was too short–ticks are already becoming mobile].[Article in German]. MMW Fortschr Med. 2008 Mar 13;150(11):12-4. PMID:18447267

Ekerfelt C, Andersson M, Olausson A, Bergström S, Hultman P. Mercury exposure as a model for deviation of cytokine responses in experimental Lyme arthritis: HgCl2 treatment decreases T helper cell type 1-like responses and arthritis severity but delays eradication of Borrelia burgdorferi in C3H/HeN mice. Clin Exp Immunol. 2007 Oct;150(1):189-97. Epub 2007 Aug 2. PMID:17672870

Emedicine Health. Lyme Disease Symptoms. http://www.emedicinehealth.com/lyme_disease/page3_em.htm#Lyme Disease Symptoms

Eskow E, Rao RV, Mordechai E. Concurrent infection of the central nervous system by Borrelia burgdorferi and Bartonella henselae: evidence for a novel tick-borne disease complex. Arch Neurol. 2001 Sep;58(9):1357-63.

Fallon BA, Levin ES, Schweitzer PJ, Hardesty D. Inflammation and central nervous system Lyme disease. Neurobiol Dis. March 2010, 37 (3) :534-41. Epub 2009 Nov 26. PMID:19944760

Fallon BA, Lipkin RB, Corbera KM, Yu S, Nobler MS, Keilp JG, Petkova E, Lisanby SH, Moeller JR, Slavov I, Van Heertum R, Mensh BD, Sackeim HA. Regional cerebral blood flow and metabolic rate in persistent Lyme encephalopathy. Arch Gen Psychiatry. 2009 May;66(5):554-63. PMID:19414715

Fallon BA, Nields JA. Lyme Disease: A Neuropsychiatric Illness. Am J Psychiatry 1994 Nov;151(11):1571-83. PMID:7943444

Feder HM Jr, Abeles M, Bernstein M, Whitaker-Worth D, Grant-Kels JM. Diagnosis, treatment, and prognosis of erythema migrans and Lyme arthritis. Clin Dermatol. 2006 Nov-Dec;24(6):509-20. PMID:17113969

Feder HM Jr , Gerber MA, Luger SW, Ryan SW. Persistence of serum antibodies to Borrelia burgdorferi in patients treated for Lyme disease. Clin Infect Dis 1992 Nov;15(5):788-93.

Feder HM Jr, Johnson BJ, O’Connell S, Shapiro ED, Steere AC, Wormser GP; Ad Hoc International Lyme Disease Group, Agger WA, Artsob H, Auwaerter P, Dumler JS, Bakken JS, Bockenstedt LK, Green J, Dattwyler RJ, Munoz J, Nadelman RB, Schwartz I, Draper T, McSweegan E, Halperin JJ, Klempner MS, Krause PJ, Mead P, Morshed M, Porwancher R, Radolf JD, Smith RP Jr, Sood S, Weinstein A, Wong SJ, Zemel L. A critical appraisal of “chronic Lyme disease”. N Engl J Med. 2007 Oct 4;357(14):1422-30. PMID:17914043

Fingerle V, Huppertz HI. [Lyme borreliosis in children. Epidemiology, diagnosis, clinical treatment, and therapy].[Article in German]. Hautarzt. 2007 Jun;58(6):541-50, quiz 551-2. PMID:17729432

Fingerle V, Wilske B. [Stage-oriented treatment of Lyme borreliosis].[Article in German]. MMW Fortschr Med. 2006 Jun 22;148(25):39-41. PMID:16859159

Finkel MJ, Halperin JJ. Nervous system Lyme neuroborreliosis revisited. Arch Neurol 1992 Jan;49(1):102-7.

Fomenko NV, Romanova EV, Mel’nikova OV, Chernousova NIa, Epikhina TI. [Detection of Borrelia DNA in the Borrelia burgdorferi sensu lato complex in the blood of patients with Ixodes tick-borne borrelios].[Article in Russian]. Klin Lab Diagn. 2006 Aug;(8):35-7. PMID:17087247

Fürst B, Glatz M, Kerl H, Müllegger RR. The impact of immunosuppression on erythema migrans. A retrospective study of clinical presentation, response to treatment and production of Borrelia antibodies in 33 patients. Clin Exp Dermatol. 2006 Jul;31(4):509-14. Erratum in Clin Exp Dermatol. 2006 Sep;31(5):751. PMID:16716151

Gheorghiev C, De Montleau F, Defuentes G. [Alcohol and epilepsy: a case report between alcohol withdrawal seizures and neuroborreliosis].[Article in French]. Brain. 2011 Jun;37(3):231-7. Epub 2010 December 3. PMID:21703439

Ghosh S, Huber BT. Clonal diversification in OspA-specific antibodies from peripheral circulation of a chronic Lyme arthritis patient. J Immunol Methods. 2007 Apr 10;321(1-2):121-34. Epub 2007 Feb 6. PMID:17307198

Ghosh S, Seward R, Costello CE, Stollar BD, Huber BT. Autoantibodies from synovial lesions in chronic, antibiotic treatment-resistant Lyme arthritis bind cytokeratin-10. J Immunol. 2006 Aug 15;177(4):2486-94. PMID:16888010

Ghosh S, Steere AC, Stollar BD, Huber BT. In situ diversification of the antibody repertoire in chronic Lyme arthritis synovium. J Immunol. 2005 Mar 1;174(5):2860-9. PMID:15728496

Ginsberg L, Kidd D. Chronic and recurrent meningitis. Pract Neurol. 2008 Dec;8(6):348-61. PMID:19015295

Girschick HJ, Morbach H, Tappe D. Treatment of Lyme borreliosis. Arthritis Res Ther. 2009;11(6):258. Epub 2009 Dec 17. PMID:20067594

Gouveia EA, Alves MF, Mantovani E, Oyafuso LK, Bonoldi VL, Yoshinari NH. Profile of patients with Baggio-Yoshinari Syndrome admitted at “Instituto de Emilio Ribas Infectologia “. Rev Inst Med Trop Sao Paulo. 2010 Dec;52(6):297-303. PMID:21225212

Grabe HJ, Spitzer C, Luedemann J, Guertler L, Kramer A, John U, Freyberger HJ, Völzke H. No association of seropositivity for anti-Borrelia IgG antibody with mental and physical complaints. Nord J Psychiatry. 2008;62(5):386-91. PMID:18752103

Grygorczuk S, Hermanowska-Szpakowicz T, Kondrusik M, Pancewicz S, Zajkowska J. [Ehrlichiosis–a disease rarely recognized in Poland].[Article in Polish]. Wiad Lek. 2004;57(9-10):456-61. PMID:15765762

Grygorczuk S, Pancewicz S, Zajkowska J, Kondrusik M, Moniuszko A. [Articular symptoms in Lyme borreliosis]. [Article in Polish]. Pol Merkur Lekarski. 2008 June: 24 (144) :542-4. PMID:18702339

Grygorczuk S, Pancewicz S, Zajkowska J, Kondrusik M, Swierzbińska R, Moniuszko A, Pawlak-Zalewska W. [Reinfection in Lyme borreliosis].[Article in Polish]. Pol Merkur Lekarski. 2008 Sep;25(147):257-9. PMID:19112844

Grygorczuk S, Zajkowska J, Panasiuk A, Kondrusik M, Chmielewski T, Swierzbińska R, Pancewicz S, Flisiak R, Tylewska-Wierzbanowska S. [Activity of the caspase-3 in the culture of peripheral blood mononuclear cells stimulated with Borrelia burgdorferi antigens].[Article in Polish]. Przegl Epidemiol. 2008;62(1):85-91. PMID:18536229

Grygorczuk S, Zajkowska J, Swierzbińska R, Pancewicz S, Kondrusik M, Hermanowska-Szpakowicz T. [Concentrations of soluble factors participating in regulation of apoptosis of lymphocyte from patients with chronic lyme arthritis (preliminary report)].[Article in Polish]. Pol Merkur Lekarski. 2006 Jan;20(115):49-52. PMID:16617735

Hagberg L, Dotevall L. Neuroborreliosis with bad reputation. This is no mystical, difficult-to-treat infection!].[Article in Swedish]. Lakartidningen. 2007 Nov 28-Dec 4;104(48):3621-2. PMID:18193671

Halperin JJ. Prolonged Lyme disease treatment: enough is enough. Neurology. 2008 Mar 25;70(13):986-7. Epub 2007 Oct 10. PMID:17928578

Halperin JJ. Lyme Disease: An Evidence-Based Approach (Advances in Molecular and Cellular Biology Series). Wallingford, Oxfordshire, UK:CABI. 2011.

Halperin JJ, Krupp LB, Golightly MG, Volkman DJ. Lyme borreliosis-associated encephalopathy. Neurology 1990 Sep;40(9):1340-3.

Halperin JJ, Logigian EL, Finkel MF, Pearl RA. Practice parameters for the diagnosis of patients with nervous system Lyme borreliosis (Lyme disease). Neurology 1996 Mar;46(3):619-27. PMID:8618656

Halperin JJ, Shapiro ED, Logigian E, Belman AL, Dotevall L, Wormser GP, Krupp L, Gronseth G, Bever CT Jr; Quality Standards Subcommittee of the American Academy of Neurology. Practice parameter: treatment of nervous system Lyme disease (an evidence-based review): report of the Quality Standards Subcommittee of the American Academy of Neurology. Neurology. 2007 Jul 3;69(1):91-102. Epub 2007 May 23. Erratum in Neurology. 2008 Apr 1;70(14):1223. PMID:17522387

Hamblin T. Is chronic lymphocytic leukemia a response to infectious agents? Leuk Res. 2006 Sep;30(9):1063-4. Epub 2006 Jan 6. PMID:16406017

Hamlen R. Lyme borreliosis: perspective of a scientist-patient. Lancet Infect Dis. 2004 Oct;4(10):603-4. PMID:15451481

Hanses F, Audebert FX, Glück T, Salzberger B, Ehrenstein BP. [Suspected borreliosis – what’s behind it?].[Article in German]. Dtsch Med Wochenschr. Aug 2011;136(33):1652-5. Epub 2011 Aug 10th PMID:21833884

Harrer T, Geissdörfer W, Schoerner C, Lang E, Helm G. Seronegative Lyme neuroborreliosis in a patient on treatment for chronic lymphatic leukemia. Infection. 2007 Apr;35(2):110-3. PMID:17401717

Hassler D, Schnauffer M, Ehrfeld H, Müller E. Disappearance of specific immune response after successful therapy of chronic Lyme borreliosis. Int J Med Microbiol. 2004 Apr;293 Suppl 37:161-4. PMID:15147000

Hausotter W. [Appraisal of Lyme borreliosis].[Article in German] Versicherungsmedizin. 2004 Mar 1;56(1):25-9. PMID:15049470

Hendrickx G, De Boeck H, Goossens A, Demanet C, Vandenplas Y. Persistent synovitis in children with Lyme arthritis: two unusual cases. An immunogenetic approach. Eur J Pediatr. 2004 Nov;163(11):646-50. Epub 2004 Jul 28. PMID:15503133

Hendrickx G, Demanet C, Vandenplas Y. Persistent synovitis in two children with Lyme arthritis linked with HLA-DRB1*1104. Eur J Pediatr. 2006 Jun;165(6):420-1. Epub 2006 Mar 4. PMID:16518608

Hodzic E, Feng S, Holden K, Freet KJ, Barthold SW. Persistence of Borrelia burgdorferi following antibiotic treatment in mice. Antimicrob Agents Chemother. 2008 May;52(5):1728-36. Epub 2008 Mar 3. PMID:18316520

Holmes KD. An appraisal of “chronic Lyme disease”. N Engl J Med. 2008 Jan 24;358(4):429; author reply 430-1. PMID:18219749

Hoppa E, Bachur R. Lyme disease update. Curr Opin Pediatr. 2007 Jun;19(3):275-80. PMID:17505186

Horneff G. [Juvenile arthritides].[Article in German]. Z Rheumatol. 2010 Oct;69(8):719-35; quiz 736-7. PMID:20798949

Hospach T, Langendörfer M, Kalle TV, Tewald F, Wirth T, Dannecker GE. Mimicry of lyme arthritis by synovial hemangioma. Rheumatol Int. 2009 Dec 16. [Epub ahead of print] PMID:20013264

Hurley RA, Taber KH. Acute and chronic Lyme disease: controversies for neuropsychiatry. J Neuropsychiatry Clin Neurosci. 2008 Winter;20(1):iv-6. PMID:18305280

Hytönen J, Hartiala P, Oksi J, Viljanen MK. Borreliosis: recent research, diagnosis, and management. Scand J Rheumatol. 2008 May-Jun;37(3):161-72. PMID:18465449

The International Lyme and Associated Diseases Society (ILADS), Evidence-based guidelines for the management of Lyme disease. Expert Rev Anti-infect Ther, 2004. 2(Suppl): p. S1-S13.

Jacomo V, Kelly PJ, Raoult D (2002). Natural history of Bartonella infections (an exception to Koch’s postulate). Clin Diagn Lab Immunol. 2002 Jan;9(1):8-18. PMID:11777823

Jakobs M, Morawietz L, Rothschenk H, Hopf T, Weiner S, Schausten H, Krukemeyer
MG, Krenn V. [Synovitis score: value of histopathological diagnostics in unclear arthritis. Case reports from rheumatological pathological practice].[Article in German]. Z Rheumatol. 2007 Dec;66(8):706-12. PMID:18000669

Jarefors S, Janefjord CK, Forsberg P, Jenmalm MC, Ekerfelt C. Decreased up-regulation of the interleukin-12Rbeta2-chain and interferon-gamma secretion and increased number of forkhead box P3-expressing cells in patients with a history of chronic Lyme borreliosis compared with asymptomatic Borrelia-exposed individuals. Clin Exp Immunol. 2007 Jan;147(1):18-27. PMID:17177959

Johnson BJ, Robbins KE, Bailey RE, Cao BL, Sviat SL, Craven RB, Mayer LW, Dennis DT. Serodiagnosis of Lyme disease: Accuracy of a two-step approach using a flagella-based ELISA and immunoblotting. J Infect Dis 1996 Aug;174(2):346-53. PMID:8699065

Johnson L, Aylward A, Stricker RB. Healthcare access and burden of care for patients with Lyme disease: a large United States survey. Health Policy. 2011 Sep;102(1):64-71. Epub 2011 Jun 14. PMID:21676482

Johnson M, Feder HM Jr. Chronic Lyme disease: a survey of Connecticut primary care physicians. J Pediatr. 2010 Dec;157(6):1025-1029. e1-2. Epub 2010 Sep 1. PMID:20813379

Kaiser R. [Clinical courses of acute and chronic neuroborreliosis following treatment with ceftriaxone].[Article in German]. Nervenarzt. 2004 Jun;75(6):553-7. PMID:15257378

Kalac M, Suvic-Krizanic V, Ostojic S, Kardum-Skelin I, Barsic B, Jaksica B. Central nervous system involvement of previously undiagnosed chronic lymphocytic leukemia in a patient with neuroborreliosis. Int J Hematol. 2007 May;85(4):323-5. PMID:17483076

Kaminsky A. Erythema figuratum. [Article in English, Spanish]. Proceedings Dermosifiliogr. 2009 Dec;100 Suppl 2:88-109. PMID:20096167

Kaplan FR, Jones-Woodward L. Lyme encephalopathy: a neuropsychological perspective. Semin Neurol 1997 Mar;17(1):31-7.

Karlsson M, Hovind-Hougen K, Svenungsson B, Stiernstedt G. Cultivation and characterization of spirochetes from cerebrospinal fluid of patients with Lyme borreliosis. J Clin Microbiol 1990 Mar;28(3):473-9.

Katchanov J, Siebert E, Klingebiel R, Endres M. Infectious vasculopathy of intracranial large- and medium-sized vessels in neurological intensive care unit: a clinical-radiological study. Neurocrit Care. 2010 Jun;12(3):369-74. PMID:20146025

Keller TL, Halperin JJ, Whitman M. PCR detection of Borrelia burgdorferi DNA in cerebrospinal fluid of Lyme neuroborreliosis patients. Neurology 1992 Jan;42(1):32-42.

Kemperman MM, Bakken JS, Kravitz GR. Dispelling the chronic Lyme disease myth. Minn Med. 2008 Jul;91(7):37-41. PMID:18714930

Kestelyn PG. An eye on inflammatory eye disease. Acta Clin Belg. 2005 Sep-Oct;60(5):270-5. PMID:16398326

Kisand KE, Prükk T, Kisand KV, Lüüs SM, Kalbe I, Uibo R. Propensity to excessive proinflammatory response in chronic Lyme borreliosis. APMIS. 2007 Feb;115(2):134-41. PMID:17295680

Kiser, K. In the Lyme light. Minn Med. 2009 Nov;92(11):10-2. PMID:20069988

Klimkiewicz Wolańska-E, Szymanska J, Bachanek T. Orofacial symptoms related to boreliosis–case report. Agric Environ Med Ann. 2010 Dec;17(2):319-21. PMID:21186776

Kohler J, Kern U, Kasper J, Rhese-Kupper B, Thoden U. Chronic central nervous system involvement in Lyme borreliosis. Neurology 1988 Jun;38(6):863-7.

Kordick DL, Breitschwerdt EB. Intraerythrocytic presence of Bartonella henselae. J Clin Microbiol. 1995 Jun;33(6):1655-6.

Kordick DL, Breitschwerdt EB. Relapsing bacteremia after blood transmission of Bartonella henselae to cats. Am J Vet Res. 1997 May;58(5):492-7.

Kordick DL, Breitschwerdt EB. Persistent infection of pets within a household with three Bartonella species. Emerg Infect Dis. 1998 Apr-Jun;4(2):325-8.

Kordick SK, Breitschwerdt EB, Hegarty BC, Southwick KL, Colitz CM, Hancock SI, Bradley JM, Rumbough R, Mcpherson JT, MacCormack JN. Coinfection with multiple tick-borne pathogens in a Walker Hound kennel in North Carolina. J Clin Microbiol. 1999 Aug;37(8):2631-8.

Krause A, Fingerle V. [Lyme borreliosis].[Article in German]. Z Rheumatol. 2009 May;68(3):239-52, quiz 253-4. PMID:19387665

Krause A, Herzer P. [Early diagnosis of Lyme arthritis].[Article in German]. Z Rheumatol. 2005 Nov;64(8):531-7. PMID:16328757

Kremer S, Holl N, Schmitt E, De Sèze J, Moser T, Dieterich JL Mann. [Imaging of non-traumatic and non-tumoral cord lesions]. [Article in French]. J Radiol. 2010 Sep;91(9 Pt 2):969-87. PMID:20814389

Kruger H, Kohlhepp W, Konig S. Follow-up of antibiotically treated and untreated neuroborreliosis. Acta Neurol Scand 1990 Jul;82(1):59-67.

Krupp LB. Lyme disease. In: Samuels MA, Feske S, eds. Office practice of neurology. London:Churchill-Livingstone, 1996; pp 383-7.

Kuenzle S, von Büdingen HC, Meier M, Harrer MD, Urich E, Becher B, Goebels N. Pathogen specificity and autoimmunity are distinct features of antigen-driven immune responses in neuroborreliosis. Infect Immun. 2007 Aug;75(8):3842-7. Epub 2007 May 21. PMID:17517881

Kuhn TS. The structures of scientific revolutions. Chicago: University Of Chicago Press; 3rd edition;1996. Summarized: http://des.emory.edu/mfp/Kuhn.html

LaFleur RL, Dant JC, Wasmoen TL, Callister SM, Jobe DA, Lovrich SD, Warner TF, Abdelmagid OR, Schell RF. Bacterin that induces anti-OspA and anti-OspC borreliacidal antibodies provides a high level of protection against canine Lyme disease. Clin Vaccine Immunol. 2009 Feb;16(2):253-9. Epub 2008 Dec 3. PMID:19052162

Lantos PM. Chronic Lyme disease: the controversies and the science. Expert Rev Anti Infect Ther. 2011 Jul;9(7):787-97. PMID:21810051

Lappin MR, Breitschwerdt E, Brewer M, Hawley J, Hegarty B, Radecki S. Prevalence of Bartonella species antibodies and Bartonella species DNA in the blood of cats with and without fever. J Feline Med Surg. 2009 Feb;11(2):141-8. Epub 2008 Aug 29.

Lee G, Xiang Z, Brannagan TH 3rd, Chin RL, Latov N. Differential gene expression in chronic inflammatory demyelinating polyneuropathy (CIDP) skin biopsies. J Neurol Sci. 2010 Mar 15;290(1-2):115-22. Epub 2009 Nov 17. PMID:19922956

Lesnicar G, Zerdoner D. Temporomandibular joint involvement caused by Borrelia Burgdorferi. J Craniomaxillofac Surg. 2007 Dec;35(8):397-400. Epub 2007 Oct 17. PMID:17942315

Leverkus M., Finner AM, Pokrywka A, Franke I, Gollnick H. Metastatic squamous cell carcinoma of the ankle in long-standing untreated acrodermatitis chronica atrophicans. Dermatology. 2008;217(3):215-8. Epub 2008 Jul 8. PMID:18607109

Liang FT, Brown EL, Wang T, Iozzo RV, Fikrig E. Protective niche for Borrelia burgdorferi to evade humoral immunity. Am J Pathol. 2004 Sep;165(3):977-85. PMID:15331421

Lins H, Wallesch CW, Wunderlich MT. Sequential analyses of neurobiochemical markers of cerebral damage in cerebrospinal fluid and serum in CNS infections. Acta Neurol Scand. 2005 Nov;112(5):303-8. PMID:16218912

Listernick R. A 17-year-old boy previously diagnosed with chronic Lyme disease. Patient complained of low-grade fevers, headaches, pharyngitis, and suspected his mother was trying to poison him. Pediatr Ann. 2004 Aug;33(8):494-8. PMID:15354601

Ljøstad U, Mygland A. [Lyme borreliosis in adults].[Article in Norwegian]. Tidsskr Nor Laegeforen. 2008 May 15;128(10):1175-8. PMID:18480867

Ljøstad U, Mygland A. Remaining complaints 1 year after treatment for acute Lyme neuroborreliosis; frequency, pattern and risk factors. Eur J Neurol. 2010 Jan;17(1):118-23. Epub 2009 Jul 23. PMID:19645771

Logigian EL. Neurologic manifestations of Lyme disease. In: Rahn QW, Evans J, eds. Lyme disease. Philadelphia:ACP, 1998; pp 89-106.

Logigian EL, Kaplan RF, Steere AC. Chronic neurologic manifestations of Lyme disease. N Engl J Med 1990 Nov;323(21):1438-44.

Lu B, PereiraPerrin M. A novel immunoprecipitation strategy identifies a unique functional mimic of the glial cell line-derived neurotrophic factor family ligands in the pathogen Trypanosoma cruzi. Infect Immun. 2008 Aug, 76 (8) :3530-8. Epub 2008 Jun 9. PMID:18541656

Lukashova LV, Karpova MR, Pirogova NP, Kiiutsina TA, Lepekhin AV, Perevozchikova TV, Faĭt EA. [Functional status of peripheral blood monocyte in patients with Ixodes tick-borne borreliosis accompanied by opisthorchiasis].[Article in Russian]. Zh Mikrobiol Epidemiol Immunobiol. 2006 Mar-Apr;(2):81-3. PMID:16758907

Maco V, Maguiña C, Tirado A, Maco V, Vidal JE. Carrion’s disease (Bartonellosis bacilliformis) confirmed by histopathology in the High Forest of Peru. Rev Inst Med Trop Sao Paulo. 2004 May-Jun;46(3):171-4. PMID:15286824

Maggi RG, Breitschwerdt EB. Isolation of bacteriophages from Bartonella vinsonii subsp. berkhoffii and the characterization of Pap31 gene sequences from bacterial and phage DNA. J Mol Microbiol Biotechnol. 2005;9(1):44-51.

Maggi RG, Breitschwerdt EB. Potential limitations of the 16S-23S rRNA intergenic region for molecular detection of Bartonella species. J Clin Microbiol. 2005 Mar;43(3):1171-6.

Maloney E. Chronic lyme disease counterpoint. Minn Med. 2008 Aug;91(8):6-7. PMID:18773702

Maloney EL. An appraisal of “chronic Lyme disease”. N Engl J Med. 2008 Jan 24;358(4):428-9; author reply 430-1. PMID:18219748

Maloney EL. Article shed no light. Minn Med. 2010 Jan;93(1):6-7. PMID:20191722

Markeljević J, Sarac H, Rados M. Tremor, seizures and psychosis as presenting symptoms in a patient with chronic Lyme neuroborreliosis (LNB). Coll Antropol. 2011 Jan;35 Suppl 1:313-8. PMID:21648354

Marques A. Chronic Lyme disease: a review. Infect Dis Clin North Am. 2008 Jun;22(2):341-60, vii-viii. PMID:18452806

Martí-Martínez S, Martín-Estefanía C, Turpín-Fenoll L, Pampliega-Pérez A, Reus-Bañuls S, García-Barragán N, Villarubia-Lor B. [Bilateral papilloedema as the initial symptom of POEMS syndrome].[Article in Spanish]. Rev Neurol. 2006 Nov 1-15;43(9):531-4. PMID:17072808

Mayer L, Merz S. An appraisal of “chronic Lyme disease”. Engl J Med. 2008 Jan 24;358(4):428; author reply 430-1. PMID:18216368

Mayo Clinic Staff. Lyme Disease Symptoms. http://www.mayoclinic.com/health/lyme-disease/DS00116/DSECTION=symptoms

McGill S, Hjelm E, Rajs J, Lindquist O, Friman G. Bartonella spp. antibodies in forensic samples from Swedish heroin addicts. Ann N Y Acad Sci. 2003 Jun;990:409-13. PMID:12860665

Mervin P. Don’t deny treatment. Minn Med. 2009 Dec;92(12):6. PMID:20092159

Michau TM, Breitschwerdt EB, Gilger BC, Davidson MG. Bartonella vinsonii subspecies berkhoffi as a possible cause of anterior uveitis and choroiditis in a dog. Vet Ophthalmol. 2003 Dec;6(4):299-304.

Michel JM, Sellal F. [“Reversible” dementia in 2011].[Article in French]. Old Geriatr Psychol neuropsychiatrist. 2011 Jun;9(2):211-25. PMID:21690030

Miklossy J. Chronic inflammation and amyloidogenesis in Alzheimer’s disease — role of Spirochetes. J Alzheimers Dis. 2008 May;13(4):381-91. PMID:18487847

Miklossy J, Kasas S, Zurn AD, McCall S, Yu S, McGeer PL. Persisting atypical and cystic forms of Borrelia burgdorferi and local inflammation in Lyme neuroborreliosis. J Neuroinflammation. 2008 Sep 25;5:40. PMID:18817547

Miklossy J, Khalili K, Gern L, Ericson RL, Darekar P, Bolle L, Hurlimann J, Paster BJ. Borrelia burgdorferi persists in the brain in chronic lyme neuroborreliosis and may be associated with Alzheimer disease. J Alzheimers Dis. 2004 Dec;6(6):639-49; discussion 673-81. PMID:15665404

Miller JC, von Lackum K, Woodman ME, Stevenson B. Detection of Borrelia burgdorferi gene expression during mammalian infection using transcriptional fusions that produce green fluorescent protein. Microb Pathog. 2006 Jul;41(1):43-7. Epub 2006 May 24. PMID:16723206

Mitty J, Margolius D. Updates and controversies in the treatment of Lyme disease. Med Health R I. 2008 Jul;91(7):219, 222-3. PMID:18705223

Moniuszko A, Czupryna P, Zajkowska J, Pancewicz SA, Grygorczuk S, Kondrusik M. [Post Lyme syndrome as a clinical problem]. [Article in Polish]. Pol Merkur Lekarski. 2009 Mar;26(153):227-30. PMID:19388538

Morales SC, Breitschwerdt EB, Washabau RJ, Matise I, Maggi RG, Duncan AW. Detection of Bartonella henselae DNA in two dogs with pyogranulomatous lymphadenitis. J Am Vet Med Assoc. 2007 Mar 1;230(5):681-5.

Mosbacher M, Elliott SP, Shehab Z, Pinnas JL, Klotz JH, Klotz SA. Cat scratch disease and arthropod vectors: more to it than a scratch? J Am Board Fam Med. 2010 Sep-Oct;23(5):685-6. PMID:20823366

Mulleger RR, Millner MM, Stanek, Spork KD. Penicillin G and ceftriaxone in the treatment of neuroborreliosis in children – a prospective study. Infection 1991 Jul-Aug;19(4):279-83.

Mygland A, Skarpaas T, Ljøstad U. Chronic polyneuropathy and Lyme disease. Eur J Neurol. 2006 Nov;13(11):1213-5. PMID:17038034

Nadelman RB, Arlen Z, Wormser GP. Life threatening complications of empiric ceftriaxone for ‘seronegative’ Lyme disease. South Med J 1991 Oct;84(10):1263-5.

Nafeev AA Klimova LV. [Clinical manifestations of neuroborreliosis in the Volga region].[Article in Russian]. Ter Arkh. 2010;82(11):68-70. PMID:21381354

Narayan K, Dail D, Li L, Cadavid D, Amrute S, Fitzgerald-Bocarsly P, Pachner AR. The nervous system as ectopic germinal center: CXCL13 and IgG in lyme neuroborreliosis. Ann Neurol. 2005 Jun;57(6):813-23. PMID:15929033

Nau R, Christian HJ, Eiffert H. Lyme disease–current state of knowledge. Dtsch Arztebl Int. 2009 Jan;106(5):72-81, 82 quiz, I. Epub 2009 Jan 30. PMID:19562015

Nigrovic LE, Thompson KM. The Lyme vaccine: a cautionary tale. Epidemiol Infect. 2007 Jan;135(1):1-8. Epub 2006 Aug 8. PMID:16893489

[No authors listed] [Differential aspects of multiple sclerosis and chronic borrelial encephalomyelitis].[Article in Russian]. Nevrol Zh Im SS Korsakova Psikhiatr. 2011;111(7):8-12. PMID:21947065

Nocton JJ, Bloom BJ, Rutledge BJ, Logigian EL, Schmid CH, Steere AC. Detection of Borrelia burgdorferi DNA by polymerase chain reaction in cerebrospinal fluid in Lyme neuroborreliosis. J Infect Dis 1996 Sep;174(3):623-7.

Nygård K, Brantsaeter AB, Mehl R. Disseminated and chronic Lyme borreliosis in Norway, 1995 – 2004. Euro Surveill. 2005 Oct;10(10):235-8. PMID:16282646

Ogrinc K, Logar M, Lotric-Furlan S, Cerar D, Ruzić-Sabljić E, Strle F. Doxycycline versus ceftriaxone for the treatment of patients with chronic Lyme borreliosis. Wien Klin Wochenschr. 2006 Nov;118(21-22):696-701. PMID:17160610

Oksi J, Nikoskelainen J, Hiekkanen H, Lauhio A, Peltomaa M, Pitkäranta A, Nyman D, Granlund H, Carlsson SA, Seppälä I, Valtonen V, Viljanen M. Duration of antibiotic treatment in disseminated Lyme borreliosis: a double-blind, randomized, placebo-controlled, multicenter clinical study. Eur J Clin Microbiol Infect Dis. 2007 Aug;26(8):571-81. PMID:17587070

Ostendorf GM. [No work disability in supposed post-borreliosis syndrome. On the decision of the OLG Saarbrücken of 19 May 2010].[Article in German].Versicherungsmedizin. 2011 Jun 1;63(2):106-7. PMID:21698949

Ostfeld RS. Lyme Disease: The Ecology of a Complex System. New York: Oxford University Press. 2011

Pachner AR. Lyme neuroborreliosis. In: Johnson RT, Griffin JW, eds. Current therapy in neurologic disease. St Louis: Mosby, 1997; pp 140-6.

Pachner AR, Delaney E. The polymerase chain reaction in the diagnosis of Lyme neuroborreliosis. Ann Neurol 1993 Oct;34(4):544-50.

Pachner AR, Duray P, Steere AC. Central nervous system manifestations of Lyme disease. Arch Neurol. 1989 Jul;46(7):790-5.

Pachner AR, Steere AC. The triad of neurologic manifestations of Lyme disease: meningitis, cranial neuritis, and radiculoneuritis. Neurology. 1985 Jan;35(1):47-53.

Pancewicz S, Popko J, Rutkowski R, Knaś M, Grygorczuk S, Guszczyn T, Bruczko M, Szajda S, Zajkowska J, Kondrusik M, Sierakowski S, Zwierz K. Activity of lysosomal exoglycosidases in serum and synovial fluid in patients with chronic Lyme and rheumatoid arthritis. Scand J Infect Dis. 2009;41(8):584-9. PMID:19513935

Papo T. [Could aspecific symptoms be related to Borrelia infection?].[Article in French]. Med Mal Infect. 2007 Jul-Aug;37(7-8):507-10. Epub 2007 Mar 13. PMID:17360137

Parish JM. Sleep-related problems in common medical conditions. Chest. 2009 Feb;135(2):563-72. PMID:19201722

Parker M, Turhan V, Aslan M, Musellim B, Hot Topic Y, Ertugrul B. [First report of three culture confirmed human Lyme cases in Turkey].[Article in Turkish]. Find Antimicrob. 2010 Jan;44(1):133-9. PMID:20455410

Persecă T, Feder A, Molnar GB. [Results of etiologic diagnosis in clinical syndrome consistent with acute and chronic borreliosis].[Article in Romanian]. Rev Med Chir Soc Med Nat Iasi. 2008 Apr-Jun;112(2):496-501. PMID:19295026

Pfister HW. [Clinical aspects of neuroborreliosis].[Article in German]. MMW Fortschr Med. 2010 Jul 1;152(25-27):31-4; quiz 35. PMID:20672660

Pfister HW, Rupprecht TA. Clinical aspects of neuroborreliosis and post-Lyme disease syndrome in adult patients. Int J Med Microbiol. 2006 May;296 Suppl 40:11-6. Epub 2006 Mar 9. PMID:16524775

Phillips SE, Burrascano JJ, Harris NS, Johnson L, Smith PV, Stricker RB. Chronic infection in ‘post-Lyme borreliosis syndrome’. Int J Epidemiol. 2005 Dec;34(6):1439-40; author reply 1440-3. Epub 2005 Nov 30. PMID:16319107

Pourel J. [Clinical diagnosis of Lyme borreliosis in case of joint and muscular presentations].[Article in French]. Med Mal Infect. 2007 Jul-Aug;37(7-8):523-31. Epub 2007 Mar 26. PMID:17368783

Przytuła L, Gińdzieńska-Sieśkiewicz E, Sierakowski S. [Diagnosis and treatment of Lyme arthritis].[Article in Polish]. Przegl Epidemiol. 2006;60 Suppl 1:125-30. PMID:16909789

Puéchal X. [Non antibiotic treatments of Lyme borreliosis].[Article in French]. Med Mal Infect. 2007 Jul-Aug;37(7-8):473-8. Epub 2007 Mar 21. PMID:17376627

Puius YA, Kalish RA. Lyme arthritis: pathogenesis, clinical presentation, and management. Infect Dis Clin North Am. 2008 Jun;22(2):289-300, vi-vii. PMID:18452802

Reik L Jr. Lyme Disease and the Nervous System. New York:Thieme Medical Publishers. 1991, pp 57-61.

Reik L Jr. Neurologic aspects of North American Lyme disease. In Lyme Disease, ed. Patricia K. Coyle, M.D. St. Louis:Mosby-Year Book Inc. 1993, pp.101-112.

Renaud I, Cachin C, Gerster JC. Good outcomes of Lyme arthritis in 24 patients in an endemic area of Switzerland. Joint Bone Spine. 2004 Jan;71(1):39-43. PMID:14769519

Reshetova GG, Zaripova TN, Titskaia EV, Moskvin VS, Udintsev SN. [Physical factors in rehabilitation treatment of patients with Ixodes tick-borne borreliosis with primary lesions of the joints].[Article in Russian]. Vopr Kurortol Fizioter Lech Fiz Kult. 2004 Nov-Dec;(6):10-3. PMID:15717529

Roche Lanquetot MO, Ader F, Durand MC, Carlier R, Defferriere H, Dinh A, Herrmann JL, Guillemot D, Perrone C, Salomon J. [Results of a prospective standardized study of 30 patients with chronic neurological and cognitive disorders after tick bites].[Article in French]. Med Mal Infect. 2008 Oct;38(10):543-8. PMID:18722064

Rolain JM, Brouqui P, Koehler JE, Maguina C, Dolan MJ, Raoult D. Recommendations for treatment of human infections caused by Bartonella species. Antimicrob Agents Chemother. 2004 Jun;48(6):1921-33. PMID:15155180

Rorat M, Kuchar E, Szenborn L, Małyszczak K. [Growing boreliosis anxiety and its reasons].[Article in Polish]. Psychiatr Pol 2010 Nov-Dec;44(6):895-904. PMID:21449171

Rossi M. [Late manifestations of Lyme borreliosis].[Article in German]. Ther Umsch. 2005 Nov;62(11):745-9. PMID:16350537

Roth J, Scheer I, Kraft S, Keitzer R, Riebel T. Uncommon synovial cysts in children. Eur J Pediatr. 2006 Mar;165(3):178-81. Epub 2005 Dec 13. PMID:16344992

Rudenko N, Golovchenko M, Růzek D, Piskunova N, Mallátová N, Grubhoffer L. Molecular detection of Borrelia bissettii DNA in serum samples from patients in the Czech Republic with suspected borreliosis. FEMS Microbiol Lett. March 2009, 292 (2) :274-81. Epub 2009 Jan 28. PMID:19187198

Samuels DS, Radolf JD, eds. Borrelia: Molecular Biology, Host Interaction and Pathogenesis. Norfolk, UK: Caister Academic Press. 2010.

Savely VR. Update on lyme disease: the hidden epidemic. Brews J Nurs. 2008 Jul-Aug;31(4):236-40. PMID:18641487

Savely V. Lyme disease: a diagnostic dilemma. Nurse Pract. 2010 Jul;35(7):44-50. PMID:20555245

Schaller J. The Diagnosis, Treatment and Prevention of Bartonella: Atypical Bartonella Treatment Failures and 40 Hypothetical Physical Exam Findings – Full Color Edition. Volume I-II. Tampa, FL:Hope Academic Press. 2008.

Schaller J. Babesia. in Encyclopedia of Plagues, Pestilence and Pandemics. Ed. J. Bryre. Westport, CT: Greenwood Press; 2008.

Schaller J. Bartonella. in Encyclopedia of Plagues, Pestilence and Pandemics. Ed. J. Bryre, Westport, CT: Greenwood Press; 2008

Schaller J. Lyme Disease. in Encyclopedia of Plagues, Pestilence and Pandemics. Ed. J. Bryre. Westport, CT: Greenwood Press; 2008

Schaller J. Babesia 2009 Supplement and Update. Tampa, FL:Hope Academic Press. 2009.

Schaller JL. Artemisin, Artesunate, Artemisinic Acid and Other Derivatives of Artemisia Used for Malaria, Babesia and Cancer. Tampa, FL: Hope Academic Press. 2006.

Schaller JL. The Health Care Professional’s Guide to the Treatment and Diagnosis of Human Babesiosis, An Extensive Review of New Human Species and Advanced Treatments. Tampa, FL: Hope Academic Press. 2006.

Schaller JL, Burkland GA. Case report: rapid and complete control of idiopathic hypereosinophilia with imatinib mesylate. MedGenMed. 2001;3(5):9.

Schaller JL, Burkland GA, Langhoff PJ. Are various Babesia species a missed cause for hypereosinophilia? A follow-up on the first reported case of imatinib mesylate for idiopathic hypereosinophilia. MedGenMed. 2007 Feb 27;9(1):38.

Schaller JL, Burkland GA, Langhoff PJ. Do bartonella infections cause agitation, panic disorder, and treatment-resistant depression? MedGenMed. 2007 Sep 13;9(3):54.

Scheffer RE, Linden S. Concurrent medical conditions with pediatric bipolar disorder. Curr Opin Psychiatry. 2007 Jul;20(4):398-401. PMID:17551356

Schnarr S, Franz JK, Krause A, Zeidler H. Infection and musculoskeletal conditions: Lyme borreliosis. Best Pract Res Clin Rheumatol. 2006 Dec;20(6):1099-118. PMID:17127199

Schutzer SE, Angel TE, Liu T, Schepmoes AA, TR Clauss, JN Adkins, DG Camp, Holland BK, Bergquist J, Coyle PK, Smith RD, Fallon BA, Natelson BH. Distinct cerebrospinal fluid proteomes differentiate post-treatment lyme disease from chronic fatigue syndrome. PLoS One. 2011 Feb 23;6(2):e17287. PMID:21383843

Schweighofer CD, Fätkenheuer G, Staib P, Hallek M, Reiser M. Lyme disease in a patient with chronic lymphocytic leukemia mimics leukemic meningeosis. Onkologie. 2007 Nov;30(11):564-6. Epub 2007 Oct 16. PMID:17992027

Science Daily (Jan 6, 2009). New Bartonella Species That Infects Humans Discovered. Available at http://www.sciencedaily.com/releases/2009/01/090106145006.htm

Shapiro ED. Tick-borne diseases. Adv Pediatr Infect Dis. 1997;13:187-218. Review.

Shapiro ED. Long-term outcomes of persons with Lyme disease. Vector Borne Zoonotic Dis. 2002 Winter;2(4):279-81.

Shapiro ED, Gerber MA. Lyme disease and facial nerve palsy. Arch Pediatr Adolesc Med. 1997 Dec;151(12):1183-4.

Sherr VT. Human babesiosis–an unrecorded reality. Absence of formal registry undermines its detection, diagnosis and treatment, suggesting need for immediate mandatory reporting. Med Hypotheses. 2004;63(4):609-15. PMID:15325004

Sherr VT. Munchausen’s syndrome by proxy and Lyme disease: medical misogyny or diagnostic mystery? Med Hypotheses. 2005;65(3):440-7. PMID:15925450

Siegel DM. Chronic arthritis in adolescence. Adolesc Med State Art Rev. 2007 May;18(1):47-61, viii. PMID:18605390

Sigal LH. Summary of the first 100 patients seen at a Lyme disease referral center. Am J Med 1990 Jun;88(6):577-83. PMID:2346158

Sigal LH. Current recommendations for the treatment of Lyme disease. Drugs 1992 May;43(5):683-99. PMID:1379147

Sigal LH. Long-term consequences of Lyme disease. In: Rahn QW, Evans J, eds. Lyme disease. Philadelphia:ACP, 1998; pp 137-53.

Sigal LH, Hassett AL. Commentary: ‘What’s in a name? That which we call a rose by any other name would smell as sweet.’ Shakespeare W. Romeo and Juliet, II, ii(47-48). Int J Epidemiol. 2005 Dec;34(6):1345-7. Epub 2005 Sep 2. PMID:16143662

Simakova AI, Popov AF, Dadalova OB. [Ixodes tick-borne borreliosis with erythema nodosum].[Article in Russian]. Med Parazitol (Mosk). 2005 Oct-Dec;(4):31-2. PMID:16445235

Sjöwall J, Carlsson A, Vaarala O, Bergström S, Ernerudh J, Forsberg P, Ekerfelt C. Innate immune responses in Lyme borreliosis: enhanced tumour necrosis factor-alpha and interleukin-12 in asymptomatic individuals in response to live spirochetes. Clin Exp Immunol. 2005 Jul;141(1):89-98. PMID:15958074

Skotarczak B. Canine ehrlichiosis. Ann Agric Environ Med. 2003;10(2):137-41. PMID:14677903

Smith HM, Reporter R, Rood MP, Linscott AJ, Mascola LM, Hogrefe W, Purcell RH. Prevalence study of antibody to ratborne pathogens and other agents among patients using a free clinic in downtown Los Angeles. J Infect Dis. 2002 Dec 1;186(11):1673-6. PMID:12447746

Smith IS, Rechlin DP. Delayed diagnosis of neuroborreliosis presenting as bell palsy and meningitis. J Am Osteopath Assoc. 2010 Aug;110(8):441-4. PMID: 20805550

Sobek V, Birkner N, Falk I, Würch A, Kirschning CJ, Wagner H, Wallich R, Lamers
MC, Simon MM. Direct Toll-like receptor 2 mediated co-stimulation of T cells in the mouse system as a basis for chronic inflammatory joint disease. Arthritis Res Ther. 2004;6(5):R433-46. Epub 2004 Jul 19. PMID:15380043

Sood SK ed. Lyme Borreliosis in Europe and North America: Epidemiology and Clinical Practice. Hoboken New Jersey: Wiley and Sons, Inc., 2011.

Speelman P, de Jongh BM, Wolfs TF, Wittenberg J; Kwaliteitsinstituut voor de
Gezondheidszorg (CBO). [Guideline ‘Lyme borreliosis’].[Article in Dutch]. Ned Tijdschr Geneeskd. 2004 Apr 3;148(14):659-63. PMID:15106316

Sréter T, Sréterné Lancz Z, Széll Z, Egyed L. [Rickettsia helvetica: an emerging tick-borne pathogen in Hungary and Europe].[Article in Hungarian]. Orv Hetil. 2005 Dec 11;146(50):2547-52. PMID:16440500

Steere AC. Musculoskeletal manifestations of Lyme disease. Am J Med. 1995 Apr 24;98(4A):44S-48S; discussion 48S-51S. Review.

Steere AC, Bartenhagen NH, Craft JE, Hutchinson GJ, Newman JH, Rahn DW, Sigal LH, Spieler PN, Stenn KS, Malawista SE. The early clinical manifestations of Lyme disease. Ann Intern Med. 1983 Jul;99(1):76-82.

Steere AC, Berardi VP, Weeks KE, Logigian EL, Ackermann R. Evaluation of the intrathecal antibody response to Borrelia burgdorferi as a diagnostic test for Lyme neuroborreliosis. J Infect Dis. 1990 Jun;161(6):1203-9.

Steere AC, Gibofsky A, Patarroyo ME, Winchester RJ, Hardin JA, Malawista SE. Chronic Lyme arthritis. Clinical and immunogenetic differentiation from rheumatoid arthritis. Ann Intern Med. 1979 Jun;90(6):896-901.

Steere AC, Malawista SE, Bartenhagen NH, Spieler PN, Newman JH, Rahn DW, Hutchinson GJ, Green J, Snydman DR, Taylor E. The clinical spectrum and treatment of Lyme disease. Yale J Biol Med. 1984 Jul-Aug;57(4):453-61.

Steere AC, Sikand VK. The presenting manifestations of Lyme disease and the outcomes of treatment. N Engl J Med. 2003 Jun 12;348(24):2472-4.

Sterman AB, Nelson S, Barclay P. Demyelinating neuropathy accompanying Lyme disease. Neurology 1982 Nov;32(11):1302-5.

Storch A, Vladimirtsev VA, Tumani H, Wellinghausen N, Haas A, Krivoshapkin VG, Ludolph AC. Viliuisk encephalomyelitis in Northeastern Siberia is not caused by Borrelia burgdorferi infection. Neurol Sci. 2008 Feb;29(1):11-4. Epub 2008 Apr 1. PMID:18379734

Stricker RB. Counterpoint: long-term antibiotic therapy improves persistent symptoms associated with lyme disease. Clin Infect Dis. 2007 Jul 15;45(2):149-57. Epub 2007 Jun 5. PMID:17578772

Stricker RB, Johnson L. Lyme disease: a turning point. Expert Rev Anti Infect Ther. 2007 Oct;5(5):759-62. PMID:17914908

Stricker RB, Johnson L. Chronic Lyme disease and the ‘Axis of Evil’. Future Microbiol. 2008 Dec;3(6):621-4. PMID:19072179

Stricker RB, Johnson L. Gender bias in chronic lyme disease. J Womens Health (Larchmt). 2009 Oct;18(10):1717-8; author reply 1719-20. PMID:19857097

Stricker RB, Johnson L. Lyme disease diagnosis and treatment: lessons from the AIDS epidemic. Minerva Med. 2010 Dec;101(6):419-25. PMID: 21196901

Stricker RB, Johnson L. Lyme disease: the next decade. Infect Drug Resist. 2011;4:1-9. Epub 2011 Jan 7. PMID:21694904

Stricker RB, Lautin A, Burrascano JJ. Lyme disease: point/counterpoint. Expert Rev Anti Infect Ther. 2005 Apr;3(2):155-65. PMID:15918774

Stricker RB, Savely VR, Motanya NC, Giclas PC. Complement split products c3a and c4a in chronic lyme disease. Scand J Immunol. 2009 Jan;69(1):64-9. PMID:19140878

Summers BA, Straubinger AF, Jacobson RH, Chang YF, Appel MJ, Straubinger RK. Histopathological studies of experimental lyme disease in the dog. J Comp Pathol. 2005 Jul;133(1):1-13. PMID:15904927

Tauber SC, Ribes S, Ebert S, Heinz T, Fingerle V, Bunkowski S, Kugelstadt D, Spreer A, Jahn O, Eiffert H, Nau R. Long-term intrathecal infusion of outer surface protein C from Borrelia burgdorferi causes axonal damage. J Neuropathol Exp Neurol. 2011 Sep;70(9):748-57. PMID:21865883

Taylor RS, Simpson IN. Review of treatment options for lyme borreliosis. J Chemother. 2005 Sep;17 Suppl 2:3-16. PMID:16315580

Telford SR III, Wormser GP. Bartonella spp. transmission by ticks not established. Emerg Infect Dis. 2010 Mar;16(3):379-84.
Tory HO, Zurakowski D, Sundel RP. Outcomes of children treated for Lyme arthritis: results of a large pediatric cohort. J Rheumatol. 2010 May;37(5):1049-55. Epub 2010 Apr 1. PMID:20360182

Treib J, Woessner R, Dobler G, Fernandez A, Hozler G, Schimrigk K. Clinical value of specific intrathecal production of antibodies. Acta virol 1997 Feb;41(1):27-30.

Tuuminen T, Hedman K, Söderlund-Venermo M, Seppälä I. Acute parvovirus B19 infection causes nonspecificity frequently in Borrelia and less often in Salmonella and Campylobacter serology, posing a problem in diagnosis of infectious arthropathy. Clin Vaccine Immunol. 2011 Jan;18(1):167-72. Epub 2010 Nov 24. PMID:21106777

Vel’gin SO, Protas II, Ponomarev VV, Drakina SA, Shcherba VV. [Clinical polymorphism of neuroborreliosis at a late stage of the disease].[Article in Russian]. Zh Nevrol Psikhiatr Im S S Korsakova. 2006;106(3):48-51. PMID:16608111

Vojdani A. Antibodies as predictors of complex autoimmune diseases and cancer. Int J Immunopathol Pharmacol. 2008 Jul-Sep;21(3):553-66. Erratum in Int J Immunopathol Pharmacol. 2008 Oct-Dec;21(4):following 1051. PMID:18831922

Volkman DJ. An appraisal of “chronic Lyme disease”. N Engl J Med. 2008 Jan 24;358(4):429; author reply 430-1. PMID:18219750

Wagner V, Zima E, Geller L, Merkely B. [Acute atrioventricular block in chronic Lyme disease].[Article in Hungarian]. Orv Hetil. 2010 Sep 26;151(39):1585-90. PMID:20840915

Wahlberg P, Nyman D. [Chronic Lyme borreliosis–fact or fiction?]. [Article in Finnish]. Duodecim. 2009;125(12):1269-76. PMID:19711595

WebMD. Lyme Disease Symptoms. http://arthritis.webmd.com/tc/lyme-disease-symptoms

Weintraub P. Cure Unknown: Inside the Lyme Epidemic. New York:Saint Martin’s Griffin, 2009.

Weissenbacher S, Ring J, Hofmann H. Gabapentin for the symptomatic treatment of chronic neuropathic pain in patients with late-stage lyme borreliosis: a pilot study. Dermatology. 2005;211(2):123-7. PMID:16088158

Weissmann G. “Chronic Lyme” and other medically unexplained syndromes. FASEB J. 2007 Feb;21(2):299-301. PMID:17267382

Widhe M, Jarefors S, Ekerfelt C, Vrethem M, Bergstrom S, Forsberg P, Ernerudh J. Borrelia-specific interferon-gamma and interleukin-4 secretion in cerebrospinal fluid and blood during Lyme borreliosis in humans: association with clinical outcome. J Infect Dis. 2004 May 15;189(10):1881-91. Epub 2004 Apr 26. PMID:15122525

Wielgat P, Pancewicz S, Hermanowska-Szpakowicz T, Kondrusik M, Zajkowska J, Grygorczuk S, Popko J, Zwierz K. [Activity of lysosomal exoglycosidases in serum of patients with chronic borrelia arthritis].[Article in Polish]. Przegl Epidemiol. 2004;58(3):451-8. PMID:15730009

Wormser GP. Treatment and prevention of Lyme disease, with emphasis on antimicrobial therapy for neuroborreliosis and vaccination. Semin Neurol. 1997 Mar;17(1):45-52. Review.

Wormser GP, Schwartz I. Antibiotic treatment of animals infected with Borrelia burgdorferi. Clin Microbiol Rev. 2009 Jul;22(3):387-95. PMID:19597005

Wormser GP, Shapiro ED. Implications of gender in chronic Lyme disease. J Womens Health (Larchmt). 2009 Jun;18(6):831-4. PMID:19514824

Zajkowska J, Czupryna P, Pancewicz SA, Kondrusik M, Moniuszko A. Acrodermatitis chronica atrophicans. Lancet Infect Dis. 2011 Oct;11(10):800. PMID:21958583

Zajkowska JM, Kondrusik M, Pancewicz SA, Grygorczuk S, Jamiołkowski J, Stalewska
J. [Comparison of test with antigen VlsE (C6) with tests with recombinant antigens in patients with Lyme borreliosis].[Article in Polish]. Pol Merkur Lekarski. 2007 Aug;23(134):95-9. PMID:18044336

Zajkowska JM, Swierzbińska R, Pancewicz SA, Kondrusik M, Hermanowska-Szpakowicz T. [Concentration of soluble CD4, CD8, CD25 receptors as well IFN-gamma and IL-4 released by lymphocyte of chronic Lyme patients cultured with 3 genotypes of Borrelia burgdorferi].[Article in Polish]. Pol Merkur Lekarski. 2004 May;16(95):447-50. PMID:15518424

Zalaudek I, Leinweber B, Kerl H, Müllegger RR. Acrodermatitis chronica atrophicans in a 15-year-old girl misdiagnosed as venous insufficiency for 6 years. 173. J Am Acad Dermatol. 2005 Jun;52(6):1091-4. PMID:15928636

Zeaiter Z, Liang Z, Raoult D. Genetic classification and differentiation of Bartonella species based on comparison of partial ftsZ gene sequences. J Clin Microbiol. 2002 Oct;40(10):3641-7. PMID:12354859

Zu-Rhein GM, Lo SC, Hulette CM, Powers JM. A novel cerebral microangiopathy with endothelial cell atypia and multifocal white matter lesions: a direct mycoplasmal infection? J Neuropathol Exp Neurol. 2007 Dec;66(12):1100-17. PMID:18090919

Lyme disease? Bah! The Very Idea!

GoAnimate.com: Lyme disease%3F Bah%21 The Very Idea%21 by sarahsmith

Like it? Create your own at GoAnimate.com. It’s free and fun!

Free Lyme Book Chapter + New Mercury Audio CDs

FREE LYME DISEASE CHAPTER! If you don’t yet own Connie Strasheim’s book, Insights Into Lyme Disease Treatment, now is a good time to explore its content. Last year we made Chapter 1 available for free online, and today, we are releasing Chapter 6 (with content on Bionoic / Photon therapy for Lyme disease)  online for free! We hope you enjoy this free, informative content.

NEW MERCURY AUDIO CD SERIES! Many people are familiar with Dr. Andrew Cutler’s books, Amalgam Illness: Diagnosis & Treatment and Hair Test Interpretation: Finding Hidden Toxicities. Now, Andrew Cutler has a new audio CD series – don’t miss it.

One man’s success using the coil machine

I like to share success stories. Here’s one man’s:


This man recommends people buy my book, Lyme Disease & Rife Machines, to learn more about this topic.

2011 Revised Rife Handbook – What’s New?

Rife Handbook by Nenah SylverThe new 2011 revised Rife Handbook is Now Available!

What’s new in the 2011 Edition? Here is a note from the author:

I am very excited to announce that a revised 2011 edition of The Rife Handbook is going to press in about two weeks. This new edition contains the same format and headings as the first hardcover edition. But careful editing, additions and revisions have brought you more pages and more information. You’ll find updated discoveries about Royal Rife’s original technology, as well as reports on advances in modern equipment. There are also two more appendices! One contains selected articles, published over the last several decades, on the use of electromagnetic fields, pulsed magnetic fields, and electrical current to treat disease and injuries. The other appendix is illustrated. It describes recently released lab studies of successful frequency experiments conducted on cell cultures, in the United States—experiments that actually bear Rife’s name!

The book is $112.50, with over 760 pages. Learn more about the book or place your order online now.


More information on the new 2011 Rife Handbook:

There are three Rife Handbooks by the same author. Published 2001: paperback, 448 pages, The Rife Handbook of Frequency Healing. Published 2009: hardcover, 760 pages, The Rife Handbook of Frequency Therapy with a Holistic Health Primer. Published 2011: hardcover, 768 pages, The Rife Handbook of Frequency Therapy and Holistic Health. This 2011 version closely follows its recent predecessor with some important updates: discoveries about how Royal Rife’s resonance therapy worked, additional treatments for cancer, Royal Rife’s original frequencies (verified by his lab notes), and two new appendices (selected published clinical trials over the past 40 years on frequency therapies, and contemporary Rife research in the United States, with newly released photos). The science, explained to the satisfaction of practitioners and researchers, is nonetheless understandable to the layperson. Chapter 4, “Frequently Asked Questions About Rife Sessions and Equipment,” explains how to select a unit that’s right for you, who can safely use this technology, how to give a Rife session to yourself (or to family, friends and pets), and offers troubleshooting tips for optimal sessions. Chapter 5, the 204-page cross-referenced Frequency Directory, contains frequencies for cancer, Crohn’s and other digestive disorders, Lyme, HIV, Multiple Sclerosis and other neurological disorders, Mycoplasma, parasites, respiratory infections, retroviruses, healing and regeneration, frequencies for Candida (tested in a Romanian laboratory), and much more. Even those who don’t own a device will find this book full of useful info: the history of medicine, differences between allopathic and holistic approaches, how synthesized drugs suppress the body’s natural function, how the FDA and drug industry conceal negative drug test results, how double-blind studies are unscientific and unethical, drug-related causes of teen violence, dangers of vaccines, life cycles of a microbe (pleomorphism) why germs alone do not cause disease, and how emotional and mental states change DNA. One chapter is devoted to complementary therapies to use with Rife therapy, including ozone, bodywork, colloidal/ionic silver, sauna detox, color, light, and information on clean food. Two stunning photo sections depict Rife’s inventions and modern frequency devices (including lasers and LEDs). Also: extensive bibliography, index, and five appendices — including an updated Resource Appendix telling where to obtain the most reliable frequency devices. All over the world, people are successfully using this therapy for a wide range of health problems, often serious. Once you read this book, you’ll be able to as well.

Specific Changes:

Chapter 1.
Updated thoughts on the rabies vaccine. Discusses the history of medicine,
differences between allopathic and holistic approaches, how synthesized
drugs suppress the body’s natural function, how the FDA and drug industry
conceal negative drug test results, how double-blind studies are
unscientific and unethical, drug-related causes of teen violence, and the
dangers of various drugs and vaccines. Along with the history of Rife’s
life and inventions.

Chapter 2.
Updated information on how Royal Rife’s resonance therapy and original
equipment actually worked. The lawsuit against the Beam Rays Corporation.
Corrections of factual errors about some of Rife’s case studies. Newly
discovered interviews with some of Rife’s colleagues, including Ben
Hubbard. How John Marsh and John Crane were entrapped and subsequently
arrested. Rife’s dilemma on being unable to patent his equipment from a
legal standpoint. New photo of original Beam Rays Corporation equipment.
Addresses the life cycles of a microbe (pleomorphism), why germs alone do
not cause disease, and how emotional and mental states change DNA.

Chapter 3.
New information on Hyperbaric Oxygen Therapy and Colloidal Silver. This
chapter is devoted to complementary therapies to use with Rife therapy,
including ozone, bodywork, colloidal/ionic silver, sauna detox, color,
light, and information on clean food.

Chapter 4.
Additional FAQs about rife therapy and equipment. Updated information on how
modern frequency devices work, including a more in-depth explanation of
scalar waves. A more in-depth explanation of duty cycle. More information
about treating Lyme Disease. Expanded and greater in-depth explanation in
the additional section, “How To Give Yourself a Rife Session.” Explains how
to select a unit, who can safely use this therapy, how to give a Rife
session to yourself (or family, friends or pets), and tips for optimal

Chapter 5.
More listings of disease conditions to reflect our increasingly complex,
disease-ridden world, along with some new frequencies and complementary
treatment protocols for certain health problems, including Morgellons. More
information on cancer treatment protocols as well as more accurate
frequencies for treating cancer. Corrections on Rife’s original frequencies
(as verified by his lab notes). The 204-pages of this chapter offers
cross-referenced Frequency Directory lists frequencies for cancer, Crohn’s
and other digestive disorders, Lyme, HIV, Multiple Sclerosis and other
neurological disorders, Mycoplasma, parasites, respiratory infections,
retroviruses, healing/regeneration, Candida, much more.

Chapter 6.
A few minor changes made to historical data.

Appendix A (Resource Appendix).
Additional listings, including a unique new plant-based air purification
unit, personal care products, and frequency and other electromedical
devices. Updated information on the most reliable electromedical and
frequency devices, and where to obtain them.

Appendix C (Healing With Electromedicine and Sound Therapies).
Brief but important changes in explanations of how various therapies work,
including a reconfiguring of all diagrams.

An extra Appendix D.
“Selected Published Studies in Electromedicine,” featuring clinical studies
since 1968 published in respected medical journals from all over the world
on the use of frequencies, EM fields, microcurrent, and pulsed magnetic
fields to treat diseases and degenerative conditions (along with, in some
cases, which frequencies were used).

An extra Appendix E.
“Rife Research in the United States,” with newly released photos of human
leukemia cells disabled by the Bare-Rife device.


The book is $112.50, with over 760 pages. Learn more about the book or place your order online now.

Not ready to order yet? Learn more about the book here or browse our other products.



Details on the 2011 ILADS Lyme Conference – Toronto, Canada

This is shaping up to be a fantastic conference. For all the details, visit the ILADS website:


The date is Friday – Sunday, October 28-30.  The venue is The Fairmont Royal York in Toronto, ON, Canada. The speaker’s list is dynamite, here are some highlights:

Conference Highlights
Friday, October 28, 2011
10:30-11:15am Borrelia Infections- diagnosis and treatment — Joseph J. Burrascano, Jr., MD
2:15pm-3:00pm Babesiosis: Updates on Diagnosis and Treatment 2011
— Richard I. Horowitz, MD
1:30pm-2:15pm Difficulties in the Laboratory Diagnosis of tick-borne and symptom-related chronic infections — Armin Schwarzbach, MD, PhD Infecto Laboratory
3:30pm-4:15pm Pregnancy and Tick-borne Diseases: Gestational Lyme — Charles Ray Jones, MD
10:30am-5:00pm Research, Writing and Publishing – Chronic Fatigue Syndrome paper discussion
— Samuel Shor, MD
Saturday, October 29, 2011
10:30-11:15am Overview of Integrative Patient Care in Tick-Borne Disease.
— Steven Bock, MD
11:15am-12:00pm Environmental Illness, Mold Exposure in Patients with Persistent Lyme
— Lisa L. Nagy, MD
11:15am-12:00pm Safety and Efficacy of Prolonged Intravenous Antibiotic Therapy for Neurologic Lyme Disease — Raphael B. Stricker, MD
1:30pm-2:15pm Food allergy, intolerance and candida. — Morton Teich, MD
2:15pm-3:00pm Can infections and immune reactions to them cause violent behavior?
— Robert C. Bransfield, MD
3:30pm-4:00 pm Morgellons Disease-the latest Lyme co- infection?
— Ginger Savely, DNP, FNP
3:30pm-4:15pm The Role of Naturopathy and Herbs in Treatment of Tick-Borne Illness.
— Susan McCamish, ND
4:00-4:30pm Advanced Techniques in Physical Diagnosis of Lyme Borreliosis Complex
— Joseph Jemsek, MD
Sunday, October 30, 2011
9:00am-9:30am Killing Borrelia burgdorferi – Is it possible?— Eva Sapi, PhD — John Drulle, MD Memorial Lectureship
10:30am-12pm Ask the Experts Panel — Robert Bransfield, MD; Daniel Cameron, MD; Joseph Jemsek, MD; Steven Bock, MD

POLL: Bartonella Treatment

One of my blind spots in previous books has been the importance of co-infections. I’m curious to hear about your experiences treating Bartonella. Has Bartonella treatment allowed your Lyme infection to be more susceptible to antibiotics, as reported by ILADS guidelines? Which Bartonella treatments did you find most effective? Have you used the commonly recommended treatments which include, Cumanda, Rifampin, Clove, HH, etc? Did your Bartonella infection become resistant to said treatments? Have electromagnetic treatments helped you? I look forwad to reading your responses below in the comments section.

My Lyme Guide Binder

My Lyme Guide is a new binder for helping patients and their friends, family and caregivers with the difficulties of managing Lyme disease & co-infections. It is written and compiled by Marjorie MacArthur Veiga, a Lyme disease sufferer, and her doctor, Sarah Fletcher, M.D.

The binder provides tools for organization, medical information and emotional support for patients and caregivers. My Lyme Guide is designed especially for parents and the children they care for, but can be used by any patient with chronic illness.

Coming soon: Website to purchase My Lyme Guide Binder

Sign up to be notified when this item is available

Elite athlete Perry Louis Fields to release Lyme book

Elite athletes know how to fight hard. If they didn’t, they wouldn’t reach the level of achievement that is possible within their sport.

That is why I am looking forward to the release of Perry Louis Fields’ new Lyme disease book, entitled The Tick Slayer. The book is slated to be released in a month or so.

Lymebook.com will be bringing this book to you when it is available, so stay tuned and sign up to be notified of it’s release here.

My Stand Up Paddle Board Race … Because Life Isn’t All About Lyme!

This is off topic. Thought I’d share a picture from my first stand up paddle board race. This sport is highly addicting, it’s like surfing but you don’t need real waves. Please test out my new blog comments system and tell me what you think. Want to try it? Done it?  Because Life Isn’t All About Lyme!  :-)     PS: That’s me in the hat. Yep, I still avoid the sun when I can! The Marshall Protocol helped me a lot and while I don’t use the drugs anymore, I still do avoid the sun. Life goes on even in long pants and a long sleeved shirt when everyone else is wearing swim trunks.


Click to enlarge

Newsletter or Book? Survey from Bryan Rosner

Note: if you are reading this in your email, it won’t display properly. Please click here to read.

Please take my short survey below.

I’ve finally reached a tipping point: I believe I have enough new information to justify a new book. It may be 2 years before a book actually happens, but there is so much new information that I feel it is my responsibility to share with readers.

I do have a question, though: I’m considering publishing a quarterly newsletter instead of a book. Which would you prefer? A newsletter or a book? The newsletter would be priced similarly to a book, perhaps $30-$40 per year. The advantage of a newsletter is that it would be updated continuosly as opposed to a static book.

Please take my short survey below:

Testimonial regarding coil machine + Bartonella drugs

I found this to be very informative and encouraging:


The story highlights a few interesting facts, facts which I have also been honing in on. Most importantly, the coil machine isn’t sufficient to deal with Bartonella by itself… powerful drugs are often needed (such as bactrim + rifampin). I think this is an important concept. Yet, man who wrote the above story has a wife and daughter who don’t have bartonella and are doing well with just the coil machine.

Just wanted to share the link.


The Bartonella Checklist

The following is reprinted with permission from Dr. James Schaller’s newest research. I also recommend Dr. Schaller’s Bartonella 2-book set.


The Bartonella Checklist

Increasing Suspicion of This Emerging Stealth Infection

1.      Insomnia [If profound fatigue this might not apply].

2.      Current anxiety that was not present at age ten.

3.      Current anxiety or depression not present at twenty years old.

4.      Knee-jerk emotional responses worse than past decades and worsening.

5.      Unusual discomfort on the soles of your feet

6.      A temperature under 98.3. A temperature under 99.0 if Lyme disease or Babesia is present

7.      Puffy tissue on insole or any part of ankles

8.      Depression

9.      Depression that is not fully controlled. [Improvement of mood is not successful in depression treatment].

10.  Gingivitis or bleeding during flossing

11.  Anxiety is poorly controlled with average dosing

12.  Depression is poorly controlled by reasonable medication trials

13.  Sleep medicines work poorly at routine dosing

14.  Rage worse with time

15.  Irritability worse with time

16.  IL-6 is very low

17.  IL-1B is very low

18.  TNF-a is in lower 10% of normal range

19.  Any skin markings or growths greater than most people

20.  Blood vessels or color on skin greater than most people

21.  Impatience > in personality when compared to ten years ago. [in a child, any can be any irritability]

22.  Cursing or hostile speech that is worse over time.

23.  One or more medical problems with unclear cause(s) and “idiopathic.”

24.  Red papules of any size.

25.  Skin tags including ones removed by dermatologist or shaved off.

26.  Unusual blood vessels of any kind including inside organs such as   bladder or intestinal walls

27.  Any skin finding in excess of 95% of most humans

28.  Skin findings showing increased blood vessels of any size

29.  Skin findings showing increased tissue formation that is increased over the flatness of surface skin.

30.  Skin showing blood vessels that are too large or too many forthe location of the blood vessels, e.g., surface thigh and calf skin with very thick surface blood vessels. Or legs, upper arms or shoulders have explosions of many fine blood vessels.

31.  Increased addictions that are more resistant to recovery than average.

32.  Increased impulsivity in contrast to past years or past decades.

33.  Burning skin sensations [this may have many causes].

34.  Itching without a clear cause and which is hard to control and remove

35.  Skin erosion without a clear cause such as a fire or chemical burn.

36.  Minor cuts or scratches which heal slowly.

37.  After a surgery, you heal very slowly.

38.  You have two tick or flea infections with two positive tick or flea borne viruses, bacteria or protozoa. [Bartonella has >30 published species in public genetic databases and has more vectors than possibly any infection in the world. Therefore, the presence of other infections such as tick borne viruses, bacteria or protozoa, should raise suspicion.

39.  Exposure to cats and dogs in excess of very incidental rare contact.

40.  The patient’s mother is suspicious for Bartonella based on newer direct and indirect testing.

41.  A sibling, father, spouse of child with any tick or flea-borne infection who shared a residence or vacation with proximity to brush.

42.  Exposure to outdoor environments with brush, wild grasses, wild streams, golf courses or woods.

43.  Outdoor expose in locations such as brush, wild grasses, wild streams or woods which happened without the use of DEET orwithout very high off- gassing essential oils on exposed skin areas.

44.  The outdoor exposures such as brush, wild grasses, wild streams or woods which occurred without permethrin on shoes, socks and all clothing. 

45.  Clear exposure to lice, fleas or ticks. [Bartonella is carried by a huge number of carriers, but for now, the % that carry Bartonella is not known. Further, the capacity to detect all new species in the vectors or in humans infected, does not exist or is not routinely available in direct testing of all human infectious Bartonella organisms in both large or specialty labs].   

46.  Stretch marks in eccentric locations, e.g., arms, upper side under armpit, around armpit or on the back.   

47.  Stretch marks filled with red, pink, purple or dark blue color.


Certainty claims or criticism about Bartonella positions without reading at least parts of 1,000 articles is confusing. How this is this possible with new Bartonella findings and understandings each month? There are also new species genetically sequences to show uniqueness almost every month in public databases. In this spirit, this scale is meant to merely increase suspicion of Bartonella, which is a super stealth infection that takes perhaps fifty days to grow out on some bacteria growth plates, and floats in the blood as it lowers fevers. It also clearly suppresses some key immune system fighting chemicals. Cure claims are made without the use of indirect testing markedly documented in superior journals, but which are not used by immensely busy clinicians working full-time.


Dr. Schaller is the author of 29 books and 27 top journal articles.  His publications address issues in at least twelve fields of medicine. He has the most recent textbooks on Bartonella, which was only recently discovered.  He has published on Bartonella under the supervision of the former editor of the Journal of the American Medical Association (JAMA), and his entries on multiple tick and flea borne infections, including Bartonella [along with Babesia and Lyme disease] were published in a respected infection textbook endorsed by the NIH Director of Infectious Disease. He has approximately six texts on tick and flea-borne infections based on his markedly unique full-time reading and study practice, which is not limited to either finite traditional or integrative progressive medicine. Since he has a medical license he has been able to sort through many truth claims by ordering lab testing. He does not follow truth claims without indirect testing laboratory proof. He has read full-time on these emerging problems for many years.


This form cannot be altered if it is printed or posted in any mannerwithout written permission. Posting in a critical evaluation is forbidden. Printing to assist in diagnostic reflections is encouraged, as long as no line is redacted or altered including these final paragraphs.  



It’s official: Lyme transmitted by pregnancy

A couple years ago, in a book I co-wrote on the Lyme-Autism connection, I cited studies that lyme disease can be transmitted via pregnancy. Well, the evidence continues to roll in.

Read the article: http://wusa9.com/news/article/157219/28/Could-Pregnant-Women-Transmit-Lyme-Disease-To-Their-Kids

Watch the video:


MMS book and DVD priced below cost

Hi everyone,

With all this talk of MMS, I thought I’d give everyone the chance to pick up the MMS book and MMS DVD at prices lower than what I paid for them. I have some inventory sitting here and it needs to move. So, for $25, you can have both the book and DVD. You can see on the below links that these items sell at retail
prices of much higher than $25.

Buy my leftover inventory, same items but much cheaper (both for $25):

($24.95) Retail link for book:
($29.95) Retail link for DVD: http://www.phaelos.com/understanding_mms.html
Total retail price for both items: $54.90

I’ve seen the MMS DVD and it is very interesting, and the book is pretty good too.


PS – I only have 12 sets left, so its first come, first served.

Happy 4th!

New study on the effectiveness of various antibiotics

Very telling article on several antibiotics and their effectiveness against Lyme disease. A couple things I found interesting: first, tinidizole is highly effective against BOTH spirochete and cyst form, and also, tinidizole doesn’t seem to work much better at high doses, so perhaps lower dosing would be able to reduce side effects while providing the desired results. http://www.canlyme.com/Sapi_et_al_2011.pdf

CCSVI information + “VIP” hormone supplementation

Hi everyone,

I thought I would share the following email I got from a friend on CCSVI, a blood flow disorder to the brain that many people are finding relief after treating (people who suffer from a broad range of problems, including Lyme and MS). Another colleague noted, though, that results from the treatment of CCSVI appear to be short lived, so personally I don’t think the treatment is ready for prime time, yet. Anyway, very interesting discussion.  If you want to learn more, here it is on Wikipedia:


Also, the below email conversation has some other useful info about hormone treatments and Shoemaker neurotoxin treatments.


Email #1:

Hey Bryan,   I had the treatment in the beginning of Nov. last year and it was a real leap forward.  My legs became much stronger, I started doing some light hiking almost every day, my balance improved(didn’t realize how wobbly i was), and I felt better mentally, including less mental fatigue.  Also I was able to handle twice the antimicrobials as before.  Unfortunately over the last few months all of those old symptoms returned again.  I wish I knew why it happened.  I heard a stat among MS patients that 43% suffer a restenosis within two years of the procedure.  I was hoping that since I have mostly eliminated my infections(per muscle testing results) that maybe I wouldn’t have a relapse.  Guess I was wrong.  I’m considering having it done again sometime this year and hopefully it sticks, because I dont know if it’s good to have the veins stretched out too many times.  There is also the option of stents but i’m not sure I want to go my whole life with stents in my neck either. Particularly because stents are designed for arteries, not veins, and so they dont tend to work as well. 

I did recently experience some improvement from following some of Dr. Shoemakers stuff.   Basically he’s discovered that many people with a biotoxin illness develop hypothalamic dysfunction.  The hypothalamus produces two important hormones, VIP and MSH which control inflammation in the body.  If the hypo has been damaged from something like lyme it cant produce enough of these hormones resulting in runaway inflammation in the body from sometimes just teeny amounts of toxin or infection.  I jumped through all the appropriate hoops, got prescribed some VIP nasal spray from Dr. Klinghardt, and within fifteen minutes of my first spray i could feel new blood rushing into my head, my hands became warm, felt better, etc..  then I herxed for a few days, and since have been tolerating treatment better than before.  Im still dealing with these frustrating cognitive issues, but adding VIP spray has moved me one step closer to wellness.

Now i’m following a biotoxin removal protocol by Patricia Kane Phd www.patriciakane.net which is focused on loading with high doses of Omega 6’s and 3’s at a 4:1 ratio and taking large doses of phosphatidylCholine for repairing and rebuilding cell membranes, as well as getting the cells to dump their accumulated toxins.  I dont know if its going to make me feel better yet but these two weeks i’ve been on the protocol i’ve been releasing a ton of toxins.  So that can only be good.  

Never in my wildest imagination did I think there could be this many different pieces to our puzzle. 

Email #2:

VIP stands for Vasoactive Intestinal Peptide.  As far as I know only one pharmacy has it, Hopkinton Drug http://www.rxandhealth.com/    Here’s Shoemaker’s new website that he’s still building out http://www.survivingmold.com/  He speaks about almost everything in reference to mold, but you can replace the word Mold with Lyme because they’re both biotoxins that trigger the same inflammatory cascade.  If you look at this link http://www.survivingmold.com/treatment/step-by-step  you will see all the steps Shoemaker says need to be addressed before VIP is used.  I dont think its very relevant to your case because you’re doing so well and most of your inflammatory biomarkers are probably in range.  I guess if any of these biomarkers are far out of the ‘normal’ range the VIP wont work, and in some cases can even make you worse.  The usual dose of the VIP is four sprays into the nose a day, alternating nostrils.  Some people scale back on the dose over the course of a few months, some people go off it completely after a few months, and some people stay at the full dose.  Basically use as needed.  The only information about the VIP spray I have is that it’s strength is 500MCG/ML  You know how different we all are but for some people in the mold community VIP spray has been a life saver. I suspect some of the veteran lyme patients have mostly eliminated their infections and are dealing with a dysfunctional hypothalamus that makes them feel like they have a raging lyme infection when really its a raging inflammatory response to a few lingering bugs and toxins. 

Shoemaker also mentions MARCoNS(multiple antibiotic resistant coagulase negative staph) which is showing up in the sinuses of nearly all chronic biotoxin sufferers.  That might be something worth looking into as well.  Because of the sinuses proximity to the hypothalamus and pituitary a sinus staph constantly poisons those glands with biotoxins.  Shoemaker uses a pharmaceutical called BEG nasal spray to eliminate it but many people develop an allergic reaction to it and so there’s a natural remedy that’s been a great alternative called Nasal Wash from www.supergoodstuff.com 

Have you addressed parasites? When you say the infections seem to keep coming back it makes me wonder if they could be living inside some parasites. I know MMS is helpful against parasites, and there are many other parasite treatments available but Dr. Klinghardt believes that to properly eliminate parasites we need to go on some strong anti-parasitics like Alinia, Albendazol, Ivermectin, etc..    Klinghardt also recently said that he’s finding if he first treats for parasites the rest of the lyme treatment goes much more quickly.  I guess Ivermectin can be obtained over the counter as a treatment for horses lol  Might be helpful to know.  

Interesting dialogue on the Marshall Protocol

Dear Dr. Shippen – Let me be of assistance.  I was on every combination of Lyme appropriate Antibiotics for over TEN YEARS I had a catheter in my chest getting another series of Antibiotics for over FOUR YEARS.  The saga of Lyme disease we all know is clearly an area of contentiousness.  However, what I know with certitude is the treatment for most of these heretofore seemingly imponderable chronic conditions in light of emerging breakthroughs in genomics and molecular biology are outdated clinical approaches.


The only thing “dangerous” to me was staying on the dead end Lyme treatment options I was on.  Thank God I was open and willing to try and LEARN a far superior approach.  Thank God for the internet that has completely change the paradigm.  Will more physicians get out of their orthodoxy and catch up to us patients?  


PBS/NJN News shot a story about Lyme and the current testing and treatment right in my home.  I was on the Front Page of Sunday “The Star Ledger” (the largest paper in the state), in addition to other major media appearances.  I know the issue and every treatment option VERY WELL I assure you.  Nothing I tried moved my clinical condition and serology even close to the what the MP so dramatically did!


My results are NOT anecdotal.  There are websites (MarshallProtocol.com; CureMyTh1.com, and others) with powerful positive (in many cases miraculous) testimony from patients from all over the world.  I ask respectfully that you and others please begin to listen  REALLY LISTEN and learn from highly informed patients.


From: Eugene ShippenSent: Monday, June 20, 2011 9:38 PM
To: Harold Smith
Subject: Re: mmi The latest on the Marshall Protocol. Latest what?


To all in this discussion, I would add the following observations, not withstanding that some members have had some positive experiences with the MP, either under treatment or through significant experience in prescribing it, such as Greg Baney, who I respect in his efforts and search for answers. What those of us that question the elements of the MP object to is the combined elements of the MP without adequate scientific support.


Some facts about the Marshall Protocol (MP):

1.     The MP has never been passed by a human experimental committee usually required before human trials are undertaken for new treatments (I have requested this information from several devotees and from Marshall himself and received no confirmation to date), nor has it undergone any controlled studies where various aspects of the protocol could be evaluated. Some patients may have benefitted, but for what reasons in the treatment – antibiotics? ARB effects? Vitamin D restriction? In any case, the treatment may take YEARS to see benefits and may require long periods of adverse (Herxheimer/immune dysregulation) reactions. Many patients cannot get through this initial phase or may never get through it. There is NO data on success/failure rates of those started on this unproven protocol. I have no doubt that some patients have been benefitted from treatment with the MP. My question would be how would they have done with antibiotics alone and would they have done better with additional vitamin D?

2.     The MP has several aspects that have potential benefits and potential side effects: 

a.     Antibiotics used in low doses cyclically or chronically (possibly beneficial as has been demonstrated by Brown and others – possibly negative effects of dysbiosis and antibiotic resistance. The use of various antibiotics such as tetracyclines and plaquenil have been used effectively for year to induce remission in various autoimmune diseases for many years. Is this the major benefit from individuals treated with the MP? I have used this treatment for years with beneficial results that occur much more quickly than those reported by Marshall’s devotees. I have concluded that this aspect is the basis for the major benefits seen in most patients. Many post-lyme patients have seen clear benefits from cyclic, diverse antibiotics in various different “protocols.”

b.     ARB with some documented anti-inflammatory effects (PPARgamma) – off label, untested at levels recommended – possibly may reduce inflammatory symptoms through several mechanisms, but potential adverse side effects at the recommended doses (as have been reported by some patients). Other ARBs, promoted by Richie Shoemaker, M. D. have better anti-inflammatory effects.

c.      ARBs have not been demonstrated to have specific “activating” or “blocking” effects on VDR. An early publication, Marshall calls it a VDR blocker; another, Marshall calls it a VDR activator. There is NO specific evidence that it has specific VDR activities despite theoretical “modeling” published by Marshall showing potential ARB linkage to VDR structure with NO data on effects.

d.     Restriction of all sources of vitamin D including sunlight exposure to the point of inducing overt well established severe deficiency of circulating levels of 25(OH)D3 – there are NO benefits ever demonstrated from this practice and clear adverse effects from sustained deficiency by all vitamin D experts published to date. There is no one other than Marshall’s group that have espoused this unproven theory. There are reports of exacerbation of symptoms in cases of Sarcoidosis with increases in vitamin D when increased 1,25D3 AND hypercalcemia are present. Interestingly, I know of NO patients that have been reported in published data of MP patients that demonstrate hypercalcemia, the hallmark of vitamin D toxicity. Many chronic infections have been shown to inhibit VDR activities that might reduce antimicrobial peptides (AMPs) that help the body fight off various infections. But vitamin D supplementation has been shown to increase AMP production, so why reduce it? Sunlight therapy has been reported for centuries to have specific healing benefits in patients with various chronic diseases, including TB, psoriasis, arthritis and autoimmune diseases and this treatment is available in the Dead Sea in Israel at the present time. This part of the MP doesn’t make common sense and my be detrimental as all studies of vitamin D deficiency have demonstrated. I have reviewed the bibliographies of many of the articles by Marshall and some of his devotees. The only source of published studies that would “support” the MP are articles written by devotees or Marshall himself in a circular bibliography fashion.

e.     The discussion of various “steroid” endocrine receptor interactions with vitamin D as a “steroid” is endocrine nonsense. Most hormones have the ability to activate or interact with similar receptors, but the actual hormones have the most effective activities with their specific receptors. To suggest that vitamin D may in some way interfere with other “steroid” receptors is an unproven theory in search of data to support the theory. Most hormones require adequate other supportive hormones for best effects. Correcting demonstrated deficiencies of various hormones, in my experience, has beneficial effects in treatment of all patients (with the possible exception of hormone dependent cancers) and I know Dr. Blaney, who prescribes the MP supports endocrine evaluation and treatment.

 Until the MP is subjected to controlled studies, it remains an unproven, (potentially dangerous?) approach to treatment of chronic diseases. Antibiotics may benefit some individuals with various chronic diseases, but the other aspects of the MP require validation before it should be promoted/supported on mmi.

Eugene Shippen, M. D. Private Practice, Shillington, PA


On Jun 20, 2011, at 2:58 PM, Harold Smith wrote:

Veny Musum and Dr. Bransfield.There are not any assumptionns but simply analytical summary of what the referenced web site details: anecdotal means one case, self reported means there is not an independent evaluator, chronic over years is evidence in the web site, and many treatments are utilized is evidenced in the web site and remitting relapsing is stated in parenthesis as my own experience of what the predominant number of chronic illness patients face.  Having read extensively about hormones and vitamin D, studied and practiced very practical medicine for over 45 years, having read extensively about the Marshall Protocol, attended Dr. Marshall presentations in which the presentation built extensively and quite illuminating on the work of others in chronic stealth infections while terminating when the audience awaited details of the evidence supporting the MP theories, and the even more in depth critical reviews of its flaws, published about Vitamin D, treated extensively Vitamin D deficient chronic illness patients, etc. , I appreciate your offer to educate me. Please do so with studies outside the cited authors of the Marshall Protocol.  From my point of view, go to CALDA web site and read the recent published LymeTimes article by Eugene Shippen MD and Harold Smith MD on Vitamin D in the 2010 publications. If you cannot access the article, I will regular mail you a copy. Although we are the authors, the references are to a multitude of other scientifically based physicians- not ourselves. I cannot find any studies on inducing vitamin D deficiency disease levels or hormone supplementation interfering with nuclear receptors in the immune system that support the confidence expressed in the “latest on the Marshall Protocol” beyond these authors. Scientific questions were presented to the MP and never answered. . There are components of the “latest” that are dangerous to patients- especiallly when an illness renders them desperate and more willing to gamble.. But I look forward to your contribution. When using MMI please include the details submitted in signature to enter MMI membership. I have included Dr. Shippen who may want to comment because I find the “latest” to have statements about hormone supplementation and hormone vitamin D that are generalizations and advice contradictedin the site’s referenced explanations about hormone supplementing This is very old ground for us and he may or may not find reasons to continue what has been a non dialogue before. Harold Smith MD private medical  practice Bloomsburg PA

Dr. Smith – You are making a whole series of assumptions about both myself, the MP and the hard science it is grounded in. Are you interested in gaining a more complete understanding?

Sent from my Verizon Wireless BlackBerry


Thank you for the  autobio info regarding Veny Musum MMI member. The link below is an anecdotal self-reporting story of recovery from a complex life altering chronic illness utilizing a complex treatment program over many years. Consequently it is likely there are both factors that made little difference, had significant impact, or were somewhat detrimental. Given that it requires months, even years to slowly steadily recover in a relapsing and remitting pattern ( my interpretation of the likely pattern), and many can be delayed, caution must be used in interpreting treatment as the one that did it all. Likewise, the concepts must make reasonable sense and be grounded in established open scientific discussion. Otherwise some conclusions may be dangerous to others. Harold Smith MD private practice Bloomsburg PA 

— On Mon, 6/20/11, Paula Carnes 



I am not a supporter of the Marshall Protocol, nor am I a believer that D levels of 5 are normal or healthy.


Paula Carnes

Biotech Investment Research

Las Vegas, NV



On Jun 20, 2011, at 5:25 AM, Harold Smith wrote:

Hello Dr. Bransfield. Perhaps I have missed a recent thread on the Marshall Protocol?  Anyone interested can find this link simply by googling their interest. For clarification purposes for myself and perhaps other MMI members, is the MMI notification of the link an endorsement that there is significant science as in MMI goals or simply a point of informaton. If one reads this site very serious questions arise: “many MP patients have kept their 25-D below 5 ng/ml for many years without any adverse effect”.  “Because hormonal supplementation can interfere with the activity of many nuclear receptors, hormonal supplementation is contraindicated”. There are some waivers to these strong recs-  It could be treated if HRT is “marked deficiency on a blood tests”. What are the deficient levels on blood tests that should or should not be treated and at what level do they match clinical symptomatology and physical exam findings?  What about a patient in the lower 40% with symptoms?  Here is the explanation: “Therefore, hormone supplementation treatment may compromise innate immune functions in a dose dependent fashion. Research is needed to validate this theory”.That last MP statment is the most or perhaps only significan statement.  There is no reasonable scientific methodology establishing not only Vit D levels of 5 ng/ml as without adverse effect or that hormone therapy is contraindicated- but also if one checks out the references there is an extremely low level of scientific methodology to nearly all the medical advice given with such confidence as this site.  What is the membership info provided by Veny Musum?  Thank you- Harold Smith MD private medical  practice Bloomsburg PA

— On Sat, 6/18/11, Robert Hi,

Below is a page explaining the Marshall protocol:

Thanks to Veny Musum for providing the link.




Robert C Bransfield, MD, DLFAPA

225 Highway 35

Red Bank, NJ 07701

Phone: 732-741-3263

Fax: 732-741-5308


Website: www.MentalHealthandIllness.com